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Functions of block of proliferation 1 during anterior development in Xenopus laevis. , Gärtner C., PLoS One. August 2, 2022; 17 (8): e0273507.
The Ribosomal Protein L5 Functions During Xenopus Anterior Development Through Apoptotic Pathways. , Schreiner C., Front Cell Dev Biol. January 1, 2022; 10 777121.
RBL1 (p107) functions as tumor suppressor in glioblastoma and small-cell pancreatic neuroendocrine carcinoma in Xenopus tropicalis. , Naert T., Oncogene. March 1, 2020; 39 (13): 2692-2706.
Dehydration stress alters the mitogen-activated-protein kinase signaling and chaperone stress response in Xenopus laevis. , Wu CW ., Comp Biochem Physiol B Biochem Mol Biol. January 1, 2020; 246-247 110461.
Innate Immune Response and Off-Target Mis-splicing Are Common Morpholino-Induced Side Effects in Xenopus. , Gentsch GE ., Dev Cell. March 12, 2018; 44 (5): 597-610.e10.
Tissue-selective effects of nucleolar stress and rDNA damage in developmental disorders. , Calo E., Nature. February 1, 2018; 554 (7690): 112-117.
The ribosome biogenesis factor Nol11 is required for optimal rDNA transcription and craniofacial development in Xenopus. , Griffin JN., PLoS Genet. March 10, 2015; 11 (3): e1005018.
A role for BMP-induced homeobox gene MIXL1 in acute myelogenous leukemia and identification of type I BMP receptor as a potential target for therapy. , Raymond A., Oncotarget. December 30, 2014; 5 (24): 12675-93.
Resting potential, oncogene-induced tumorigenesis, and metastasis: the bioelectric basis of cancer in vivo. , Lobikin M., Phys Biol. December 1, 2012; 9 (6): 065002.
Peter Pan functions independently of its role in ribosome biogenesis during early eye and craniofacial cartilage development in Xenopus laevis. , Bugner V., Development. June 1, 2011; 138 (11): 2369-78.
Claudin-like protein 24 interacts with the VEGFR-2 and VEGFR-3 pathways and regulates lymphatic vessel development. , Saharinen P., Genes Dev. May 1, 2010; 24 (9): 875-80.
A functional screen for genes involved in Xenopus pronephros development. , Kyuno J ., Mech Dev. July 1, 2008; 125 (7): 571-86.
Peptides from the amino terminal mdm-2-binding domain of p53, designed from conformational analysis, are selectively cytotoxic to transformed cells. , Kanovsky M., Proc Natl Acad Sci U S A. October 23, 2001; 98 (22): 12438-43.
A novel p53 mutant in human breast cancer revealed by multiple SSCP analysis. , Nigro V., Cancer Lett. April 29, 1994; 79 (1): 73-5.