Papers associated with non-neural ectoderm (and ins)

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	Neonatal diabetes caused by a homozygous KCNJ11 mutation demonstrates that tiny changes in ATP sensitivity markedly affect diabetes risk., Vedovato N., Diabetologia. July 1, 2016; 59 (7): 1430-1436.
	Sulfonylureas suppress the stimulatory action of Mg-nucleotides on Kir6.2/SUR1 but not Kir6.2/SUR2A KATP channels: a mechanistic study., Proks P., J Gen Physiol. November 1, 2014; 144 (5): 469-86.
	Disease progression and search for monogenic diabetes among children with new onset type 1 diabetes negative for ICA, GAD- and IA-2 Antibodies., Pörksen S., BMC Endocr Disord. September 23, 2010; 10 16.
	A novel, rapid, inhibitory effect of insulin on alpha1beta2gamma2s gamma-aminobutyric acid type A receptors., Williams DB., Neurosci Lett. September 26, 2008; 443 (1): 27-31.
	Relapsing diabetes can result from moderately activating mutations in KCNJ11., Gloyn AL., Hum Mol Genet. April 1, 2005; 14 (7): 925-34.
	Toward tissue-selective pancreatic B-cells KATP channel openers belonging to 3-alkylamino-7-halo-4H-1,2,4-benzothiadiazine 1,1-dioxides., de Tullio P., J Med Chem. July 17, 2003; 46 (15): 3342-53.
	Inhibition of recombinant K(ATP) channels by the antidiabetic agents midaglizole, LY397364 and LY389382., Proks P., Eur J Pharmacol. September 27, 2002; 452 (1): 11-9.
	The small muscle-specific protein Csl modifies cell shape and promotes myocyte fusion in an insulin-like growth factor 1-dependent manner., Palmer S., J Cell Biol. May 28, 2001; 153 (5): 985-98.
	The antimalarial agent mefloquine inhibits ATP-sensitive K-channels., Gribble FM., Br J Pharmacol. October 1, 2000; 131 (4): 756-60.

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