???pagination.result.count???
???pagination.result.page???
1
High-throughput analysis reveals novel maternal germline RNAs crucial for primordial germ cell preservation and proper migration. , Owens DA ., Development. January 15, 2017; 144 (2): 292-304.
SmShb, the SH2-Containing Adaptor Protein B of Schistosoma mansoni Regulates Venus Kinase Receptor Signaling Pathways. , Morel M., PLoS One. January 1, 2016; 11 (9): e0163283.
Basic Properties of the p38 Signaling Pathway in Response to Hyperosmotic Shock. , Ben Messaoud N., PLoS One. September 1, 2015; 10 (9): e0135249.
Venus kinase receptors control reproduction in the platyhelminth parasite Schistosoma mansoni. , Vanderstraete M., PLoS Pathog. May 29, 2014; 10 (5): e1004138.
Single blastomere expression profiling of Xenopus laevis embryos of 8 to 32-cells reveals developmental asymmetry. , Flachsova M., Sci Rep. January 1, 2013; 3 2278.
Cortical rotation and messenger RNA localization in Xenopus axis formation. , Houston DW ., Wiley Interdiscip Rev Dev Biol. January 1, 2012; 1 (3): 371-88.
The functions of maternal Dishevelled 2 and 3 in the early Xenopus embryo. , Tadjuidje E ., Dev Dyn. July 1, 2011; 240 (7): 1727-36.
Unfertilized Xenopus eggs die by Bad-dependent apoptosis under the control of Cdk1 and JNK. , Du Pasquier D., PLoS One. January 1, 2011; 6 (8): e23672.
The extracellular signal-regulated kinase-mitogen-activated protein kinase pathway phosphorylates and targets Cdc25A for SCF beta-TrCP-dependent degradation for cell cycle arrest. , Isoda M., Mol Biol Cell. April 1, 2009; 20 (8): 2186-95.
Jun NH2-terminal kinase ( JNK) prevents nuclear beta-catenin accumulation and regulates axis formation in Xenopus embryos. , Liao G., Proc Natl Acad Sci U S A. October 31, 2006; 103 (44): 16313-8.
Oncogenic Met receptor induces cell-cycle progression in Xenopus oocytes independent of direct Grb2 and Shc binding or Mos synthesis, but requires phosphatidylinositol 3-kinase and Raf signaling. , Mood K., J Cell Physiol. April 1, 2006; 207 (1): 271-85.
Functional interactions of Raf and MEK with Jun-N-terminal kinase ( JNK) result in a positive feedback loop on the oncogenic Ras signaling pathway. , Adler V., Biochemistry. August 16, 2005; 44 (32): 10784-95.
ERK2 is required for FGF1-induced JNK1 phosphorylation in Xenopus oocyte expressing FGF receptor 1. , Browaeys-Poly E., Biochim Biophys Acta. March 22, 2005; 1743 (1-2): 1-4.
FGF receptor phosphotyrosine 766 is a target for Grb14 to inhibit MDA-MB-231 human breast cancer cell signaling. , Cailliau K., Anticancer Res. January 1, 2005; 25 (6B): 3877-82.
Contribution of JNK, Mek, Mos and PI-3K signaling to GVBD in Xenopus oocytes. , Mood K., Cell Signal. May 1, 2004; 16 (5): 631-42.
An effector peptide from glutathione-S-transferase-pi strongly and selectively blocks mitotic signaling by oncogenic ras- p21. , Chie L., Protein J. April 1, 2004; 23 (3): 235-8.
Roles of PDGF in animal development. , Hoch RV., Development. October 1, 2003; 130 (20): 4769-84.
Role of JNK in hypertonic activation of Cl(-)-dependent Na(+)/H(+) exchange in Xenopus oocytes. , Goss GG., Am J Physiol Cell Physiol. December 1, 2001; 281 (6): C1978-90.
Differences in patterns of activation of MAP kinases induced by oncogenic ras- p21 and insulin in oocytes. , Ranginwale M., Exp Cell Res. September 10, 2001; 269 (1): 162-9.
Bistability in the JNK cascade. , Bagowski CP., Curr Biol. August 7, 2001; 11 (15): 1176-82.
c- Jun N-terminal kinase activation in Xenopus laevis eggs and embryos. A possible non-genomic role for the JNK signaling pathway. , Bagowski CP., J Biol Chem. January 12, 2001; 276 (2): 1459-65.
Induction of oocyte maturation by jun-N-terminal kinase ( JNK) on the oncogenic ras- p21 pathway is dependent on the raf-MEK signal transduction pathway. , Chie L., Cancer Chemother Pharmacol. January 1, 2000; 45 (6): 441-9.
Glutathione-S-Transferase as a selective inhibitor of oncogenic ras- p21-induced mitogenic signaling through blockade of activation of jun by jun-N-terminal kinase. , Villafania A., Ann Clin Lab Sci. January 1, 2000; 30 (1): 57-64.
Selective inhibition of oncogenic ras- p21 in vivo by agents that block its interaction with jun-N-kinase ( JNK) and jun proteins. Implications for the design of selective chemotherapeutic agents. , Amar S., Cancer Chemother Pharmacol. January 1, 1997; 41 (1): 79-85.
Activation of mitogen-activated protein kinase cascades by p21-activated protein kinases in cell-free extracts of Xenopus oocytes. , Polverino A., J Biol Chem. November 3, 1995; 270 (44): 26067-70.