???pagination.result.count???
???pagination.result.page???
1
WIN55,212-2, a Dual Modulator of Cannabinoid Receptors and G Protein-Coupled Inward Rectifier Potassium Channels. , An D., Biomedicines. April 28, 2021; 9 (5):
Identification of a unique endoplasmic retention motif in the Xenopus GIRK5 channel and its contribution to oocyte maturation. , Rangel-Garcia CI., FEBS Open Bio. April 1, 2021; 11 (4): 1093-1108.
A Collision Coupling Model Governs the Activation of Neuronal GIRK1/2 Channels by Muscarinic-2 Receptors. , Berlin S., Front Pharmacol. April 7, 2020; 11 1216.
Mutual action by Gγ and Gβ for optimal activation of GIRK channels in a channel subunit-specific manner. , Tabak G., Sci Rep. January 24, 2019; 9 (1): 508.
A Quantitative Model of the GIRK1/2 Channel Reveals That Its Basal and Evoked Activities Are Controlled by Unequal Stoichiometry of Gα and Gβγ. , Yakubovich D., PLoS Comput Biol. November 6, 2015; 11 (11): e1004598.
Functional diversity of voltage-sensing phosphatases in two urodele amphibians. , Mutua J., Physiol Rep. July 16, 2014; 2 (7):
Conformational changes underlying pore dilation in the cytoplasmic domain of mammalian inward rectifier K+ channels. , Inanobe A., PLoS One. January 1, 2013; 8 (11): e79844.
Inhibition of g protein-activated inwardly rectifying k channels by phencyclidine. , Kobayashi T., Curr Neuropharmacol. March 1, 2011; 9 (1): 244-6.
Inhibition of G protein-activated inwardly rectifying K+ channels by different classes of antidepressants. , Kobayashi T., PLoS One. January 1, 2011; 6 (12): e28208.
Intron 4 containing novel GABAB1 isoforms impair GABAB receptor function. , Lee C , Lee C ., PLoS One. November 16, 2010; 5 (11): e14044.
Inhibition of G-protein-activated inwardly rectifying K+ channels by the selective norepinephrine reuptake inhibitors atomoxetine and reboxetine. , Kobayashi T., Neuropsychopharmacology. June 1, 2010; 35 (7): 1560-9.
Pregnenolone sulfate potentiates the inwardly rectifying K channel Kir2.3. , Kobayashi T., PLoS One. July 21, 2009; 4 (7): e6311.
Inhibition by cocaine of G protein-activated inwardly rectifying K+ channels expressed in Xenopus oocytes. , Kobayashi T., Toxicol In Vitro. June 1, 2007; 21 (4): 656-64.
Inhibition of G protein-activated inwardly rectifying K+ channels by the antidepressant paroxetine. , Kobayashi T., J Pharmacol Sci. November 1, 2006; 102 (3): 278-87.
Inhibition of G protein-activated inwardly rectifying K+ channels by ifenprodil. , Kobayashi T., Neuropsychopharmacology. March 1, 2006; 31 (3): 516-24.
Modulators of G protein-activated inwardly rectifying K+ channels: potentially therapeutic agents for addictive drug users. , Kobayashi T., Ann N Y Acad Sci. October 1, 2004; 1025 590-4.
Inhibition of G protein-activated inwardly rectifying K+ channels by fluoxetine (Prozac). , Kobayashi T., Br J Pharmacol. March 1, 2003; 138 (6): 1119-28.
Heterologous facilitation of G protein-activated K(+) channels by beta-adrenergic stimulation via cAMP-dependent protein kinase. , Müllner C., J Gen Physiol. May 1, 2000; 115 (5): 547-58.
Inhibition by various antipsychotic drugs of the G-protein-activated inwardly rectifying K(+) ( GIRK) channels expressed in xenopus oocytes. , Kobayashi T., Br J Pharmacol. April 1, 2000; 129 (8): 1716-22.
Positive and negative coupling of the metabotropic glutamate receptors to a G protein-activated K+ channel, GIRK, in Xenopus oocytes. , Sharon D., J Gen Physiol. April 1, 1997; 109 (4): 477-90.
Specific regions of heteromeric subunits involved in enhancement of G protein-gated K+ channel activity. , Chan KW., J Biol Chem. March 7, 1997; 272 (10): 6548-55.
Dominant negative chimeras provide evidence for homo and heteromultimeric assembly of inward rectifier K+ channel proteins via their N-terminal end. , Fink M., FEBS Lett. January 2, 1996; 378 (1): 64-8.
Heterologous multimeric assembly is essential for K+ channel activity of neuronal and cardiac G-protein-activated inward rectifiers. , Duprat F., Biochem Biophys Res Commun. July 17, 1995; 212 (2): 657-63.