???pagination.result.count???
???pagination.result.page???
1
Inhibition of DNA replication initiation by silver nanoclusters. , Tao Y., Nucleic Acids Res. May 21, 2021; 49 (9): 5074-5083.
RPA-coated single-stranded DNA promotes the ETAA1-dependent activation of ATR. , Lyu K., Cell Cycle. April 1, 2019; 18 (8): 898-913.
APE2 promotes DNA damage response pathway from a single-strand break. , Lin Y., Nucleic Acids Res. March 16, 2018; 46 (5): 2479-2494.
RNAs coordinate nuclear envelope assembly and DNA replication through ELYS recruitment to chromatin. , Aze A., Nat Commun. December 14, 2017; 8 (1): 2130.
Smarcal1-Mediated Fork Reversal Triggers Mre11-Dependent Degradation of Nascent DNA in the Absence of Brca2 and Stable Rad51 Nucleofilaments. , Kolinjivadi AM., Mol Cell. September 7, 2017; 67 (5): 867-881.e7.
Molecular basis for PrimPol recruitment to replication forks by RPA. , Guilliam TA., Nat Commun. May 23, 2017; 8 15222.
Xenopus egg extract to study regulation of genome-wide and locus-specific DNA replication. , Raspelli E., Genesis. January 1, 2017; 55 (1-2):
The mechanism of DNA replication termination in vertebrates. , Dewar JM ., Nature. September 17, 2015; 525 (7569): 345-50.
RAD18 Is a Maternal Limiting Factor Silencing the UV-Dependent DNA Damage Checkpoint in Xenopus Embryos. , Kermi C., Dev Cell. August 10, 2015; 34 (3): 364-72.
DNA polymerase κ-dependent DNA synthesis at stalled replication forks is important for CHK1 activation. , Bétous R., EMBO J. July 31, 2013; 32 (15): 2172-85.
The Mre11- Rad50- Nbs1 (MRN) complex has a specific role in the activation of Chk1 in response to stalled replication forks. , Lee J ., Mol Biol Cell. May 1, 2013; 24 (9): 1343-53.
SUMO2/3 modification of cyclin E contributes to the control of replication origin firing. , Bonne-Andrea C., Nat Commun. January 1, 2013; 4 1850.
Role of replication protein A as sensor in activation of the S-phase checkpoint in Xenopus egg extracts. , Recolin B., Nucleic Acids Res. April 1, 2012; 40 (8): 3431-42.
Xenopus laevis Ctc1- Stn1- Ten1 (xCST) protein complex is involved in priming DNA synthesis on single-stranded DNA template in Xenopus egg extract. , Nakaoka H., J Biol Chem. January 2, 2012; 287 (1): 619-627.
Cdk1 uncouples CtIP-dependent resection and Rad51 filament formation during M-phase double-strand break repair. , Peterson SE., J Cell Biol. September 5, 2011; 194 (5): 705-20.
Replication initiation complex formation in the absence of nuclear function in Xenopus. , Krasinska L., Nucleic Acids Res. April 1, 2009; 37 (7): 2238-48.
A hypophosphorylated form of RPA34 is a specific component of pre-replication centers. , Françon P., J Cell Sci. October 1, 2004; 117 (Pt 21): 4909-20.
Absence of BLM leads to accumulation of chromosomal DNA breaks during both unperturbed and disrupted S phases. , Li W ., J Cell Biol. June 21, 2004; 165 (6): 801-12.
The role of Cdc6 in ensuring complete genome licensing and S phase checkpoint activation. , Oehlmann M., J Cell Biol. April 26, 2004; 165 (2): 181-90.
The Xenopus Xmus101 protein is required for the recruitment of Cdc45 to origins of DNA replication. , Van Hatten RA., J Cell Biol. November 25, 2002; 159 (4): 541-7.
DNA double-strand breaks induce formation of RP-A/Ku foci on in vitro reconstituted Xenopus sperm nuclei. , Grandi P., J Cell Sci. September 1, 2001; 114 (Pt 18): 3345-57.
Evidence for a replication function of FFA-1, the Xenopus orthologue of Werner syndrome protein. , Chen CY ., J Cell Biol. March 5, 2001; 152 (5): 985-96.
Ultrastructural characterization of RPA-containing domains in nuclei assembled in Xenopus egg extracts. , Eltsov M., J Struct Biol. April 1, 2000; 129 (2-3): 211-7.
A functional analysis of p53 during early development of Xenopus laevis. , Amariglio F., Oncogene. October 1, 1997; 15 (18): 2191-9.
Nuclear proteins of quiescent Xenopus laevis cells inhibit DNA replication in intact and permeabilized nuclei. , Fang J., J Cell Biol. June 1, 1996; 133 (5): 955-69.
An analysis of the regulation of DNA synthesis by cdk2, Cip1, and licensing factor. , Yan H., J Cell Biol. April 1, 1995; 129 (1): 1-15.