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Summary Anatomy Item Literature (1109) Expression Attributions Wiki
XB-ANAT-630

Papers associated with spermatozoon (and mre11)

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POLθ prevents MRE11-NBS1-CtIP-dependent fork breakage in the absence of BRCA2/RAD51 by filling lagging-strand gaps., Mann A., Mol Cell. November 17, 2022; 82 (22): 4218-4231.e8.                              


RPA-coated single-stranded DNA promotes the ETAA1-dependent activation of ATR., Lyu K., Cell Cycle. April 1, 2019; 18 (8): 898-913.              


Fanconi-Anemia-Associated Mutations Destabilize RAD51 Filaments and Impair Replication Fork Protection., Zadorozhny K., Cell Rep. October 10, 2017; 21 (2): 333-340.                


Smarcal1-Mediated Fork Reversal Triggers Mre11-Dependent Degradation of Nascent DNA in the Absence of Brca2 and Stable Rad51 Nucleofilaments., Kolinjivadi AM., Mol Cell. September 7, 2017; 67 (5): 867-881.e7.                


Xenopus Mcm10 is a CDK-substrate required for replication fork stability., Chadha GS., Cell Cycle. August 17, 2016; 15 (16): 2183-2195.            


The Mre11-Rad50-Nbs1 (MRN) complex has a specific role in the activation of Chk1 in response to stalled replication forks., Lee J., Mol Biol Cell. May 1, 2013; 24 (9): 1343-53.          


RAD51- and MRE11-dependent reassembly of uncoupled CMG helicase complex at collapsed replication forks., Hashimoto Y., Nat Struct Mol Biol. December 4, 2011; 19 (1): 17-24.          


Cdk1 uncouples CtIP-dependent resection and Rad51 filament formation during M-phase double-strand break repair., Peterson SE., J Cell Biol. September 5, 2011; 194 (5): 705-20.              


Rad51 protects nascent DNA from Mre11-dependent degradation and promotes continuous DNA synthesis., Hashimoto Y., Nat Struct Mol Biol. November 1, 2010; 17 (11): 1305-11.          

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