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Summary Anatomy Item Literature (565) Expression Attributions Wiki
XB-ANAT-64

Papers associated with myocardium (and kcne1)

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Virus-Host Interactions of Enteroviruses and Parvovirus B19 in Myocarditis., Ho HT., Cell Physiol Biochem. November 18, 2021; 55 (6): 679-703.        


4,4'-Diisothiocyanato-2,2'-Stilbenedisulfonic Acid (DIDS) Modulates the Activity of KCNQ1/KCNE1 Channels by an Interaction with the Central Pore Region., Bollmann E., Cell Physiol Biochem. April 8, 2020; 54 (2): 321-332.


Polyunsaturated fatty acid analogues differentially affect cardiac NaV, CaV, and KV channels through unique mechanisms., Bohannon BM., Elife. March 24, 2020; 9                                                               


Molecular cloning and functional expression of the K + channel K V 7.1 and the regulatory subunit KCNE1 from equine myocardium., Pedersen PJ., Res Vet Sci. August 1, 2017; 113 79-86.


The residue I257 at S4-S5 linker in KCNQ1 determines KCNQ1/KCNE1 channel sensitivity to 1-alkanols., Xie C., Acta Pharmacol Sin. January 1, 2016; 37 (1): 124-33.          


Polyunsaturated fatty acid analogs act antiarrhythmically on the cardiac IKs channel., Liin SI., Proc Natl Acad Sci U S A. May 5, 2015; 112 (18): 5714-9.          


Intracellular ATP binding is required to activate the slowly activating K+ channel I(Ks)., Li Y., Proc Natl Acad Sci U S A. November 19, 2013; 110 (47): 18922-7.


Extracellular potassium inhibits Kv7.1 potassium channels by stabilizing an inactivated state., Larsen AP., Biophys J. August 17, 2011; 101 (4): 818-27.


Inhibition of the heterotetrameric K+ channel KCNQ1/KCNE1 by the AMP-activated protein kinase., Alesutan I., Mol Membr Biol. February 1, 2011; 28 (2): 79-89.


PKC activation and PIP(2) depletion underlie biphasic regulation of IKs by Gq-coupled receptors., Matavel A., J Mol Cell Cardiol. May 1, 2009; 46 (5): 704-12.


KCNE2 is colocalized with KCNQ1 and KCNE1 in cardiac myocytes and may function as a negative modulator of I(Ks) current amplitude in the heart., Wu DM., Heart Rhythm. December 1, 2006; 3 (12): 1469-80.


Skipping of Exon 1 in the KCNQ1 gene causes Jervell and Lange-Nielsen syndrome., Zehelein J., J Biol Chem. November 17, 2006; 281 (46): 35397-403.


Frequency-dependent modulation of KCNQ1 and HERG1 potassium channels., Diness TG., Biochem Biophys Res Commun. May 19, 2006; 343 (4): 1224-33.


Association of the serum and glucocorticoid regulated kinase (sgk1) gene with QT interval., Busjahn A., Cell Physiol Biochem. January 1, 2004; 14 (3): 135-42.


Human beta(3)-adrenoreceptors couple to KvLQT1/MinK potassium channels in Xenopus oocytes via protein kinase C phosphorylation of the KvLQT1 protein., Kathöfer S., Naunyn Schmiedebergs Arch Pharmacol. August 1, 2003; 368 (2): 119-26.


KCNQ1 channels sense small changes in cell volume., Grunnet M., J Physiol. June 1, 2003; 549 (Pt 2): 419-27.


Regulation of KCNE1-dependent K(+) current by the serum and glucocorticoid-inducible kinase (SGK) isoforms., Embark HM., Pflugers Arch. February 1, 2003; 445 (5): 601-6.


Inhibitory effect of the class III antiarrhythmic drug nifekalant on HERG channels: mode of action., Kushida S., Eur J Pharmacol. December 13, 2002; 457 (1): 19-27.


Inhibition of cardiac potassium currents by pentobarbital., Bachmann A., Naunyn Schmiedebergs Arch Pharmacol. January 1, 2002; 365 (1): 29-37.


Chromanol 293B, a blocker of the slow delayed rectifier K+ current (IKs), inhibits the CFTR Cl- current., Bachmann A., Naunyn Schmiedebergs Arch Pharmacol. June 1, 2001; 363 (6): 590-6.


Blocking effects of the antiarrhythmic drug propafenone on the HERG potassium channel., Mergenthaler J., Naunyn Schmiedebergs Arch Pharmacol. April 1, 2001; 363 (4): 472-80.


Short- and long-term effects of amiodarone on the two components of cardiac delayed rectifier K(+) current., Kamiya K., Circulation. March 6, 2001; 103 (9): 1317-24.


Inhibition of IKs channels by HMR 1556., Gögelein H., Naunyn Schmiedebergs Arch Pharmacol. December 1, 2000; 362 (6): 480-8.


Functional coupling of human beta 3-adrenoreceptors to the KvLQT1/MinK potassium channel., Kathöfer S., J Biol Chem. September 1, 2000; 275 (35): 26743-7.


Norpropoxyphene-induced cardiotoxicity is associated with changes in ion-selectivity and gating of HERG currents., Ulens C., Cardiovasc Res. December 1, 1999; 44 (3): 568-78.


Independent and exclusive modulation of cardiac delayed rectifying K+ current by protein kinase C and protein kinase A., Lo CF., Circ Res. November 16, 1998; 83 (10): 995-1002.


Activation and inactivation of homomeric KvLQT1 potassium channels., Pusch M., Biophys J. August 1, 1998; 75 (2): 785-92.


A novel benzodiazepine that activates cardiac slow delayed rectifier K+ currents., Salata JJ., Mol Pharmacol. July 1, 1998; 54 (1): 220-30.


Effects of the chromanol 293B, a selective blocker of the slow, component of the delayed rectifier K+ current, on repolarization in human and guinea pig ventricular myocytes., Bosch RF., Cardiovasc Res. May 1, 1998; 38 (2): 441-50.


Blockade of HERG channels by the class III antiarrhythmic azimilide: mode of action., Busch AE., Br J Pharmacol. January 1, 1998; 123 (1): 23-30.


Suppression of slow delayed rectifier current by a truncated isoform of KvLQT1 cloned from normal human heart., Jiang M., J Biol Chem. September 26, 1997; 272 (39): 24109-12.


KVLQT channels are inhibited by the K+ channel blocker 293B., Bleich M., Pflugers Arch. August 1, 1997; 434 (4): 499-501.


A minK-HERG complex regulates the cardiac potassium current I(Kr)., McDonald TV., Nature. July 17, 1997; 388 (6639): 289-92.


Coassembly of K(V)LQT1 and minK (IsK) proteins to form cardiac I(Ks) potassium channel., Sanguinetti MC., Nature. November 7, 1996; 384 (6604): 80-3.


Inhibition of IKs in guinea pig cardiac myocytes and guinea pig IsK channels by the chromanol 293B., Busch AE., Pflugers Arch. October 1, 1996; 432 (6): 1094-6.


The novel class III antiarrhythmics NE-10064 and NE-10133 inhibit IsK channels expressed in Xenopus oocytes and IKs in guinea pig cardiac myocytes., Busch AE., Biochem Biophys Res Commun. July 15, 1994; 202 (1): 265-70.


K+ currents expressed from the guinea pig cardiac IsK protein are enhanced by activators of protein kinase C., Zhang ZJ., Proc Natl Acad Sci U S A. March 1, 1994; 91 (5): 1766-70.


The min K channel underlies the cardiac potassium current IKs and mediates species-specific responses to protein kinase C., Varnum MD., Proc Natl Acad Sci U S A. December 15, 1993; 90 (24): 11528-32.


Effects of [Ca2+]i and temperature on minK channels expressed in Xenopus oocytes., Busch AE., FEBS Lett. November 15, 1993; 334 (2): 221-4.

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