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Summary Anatomy Item Literature (421) Expression Attributions Wiki
XB-ANAT-682

Papers associated with acellular anatomical structure (and ache)

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Two novel mutations in the COLQ gene cause endplate acetylcholinesterase deficiency., Ishigaki K., Neuromuscul Disord. March 1, 2003; 13 (3): 236-44.


PRiMA: the membrane anchor of acetylcholinesterase in the brain., Perrier AL., Neuron. January 17, 2002; 33 (2): 275-85.


Acetylcholinesterase clustering at the neuromuscular junction involves perlecan and dystroglycan., Peng HB., J Cell Biol. May 17, 1999; 145 (4): 911-21.                  


Former neuritic pathways containing endogenous neural agrin have high synaptogenic activity., Cohen MW., Dev Biol. February 1, 1995; 167 (2): 458-68.              


Cellular and secreted forms of acetylcholinesterase in mouse muscle cultures., Rubin LL., J Neurochem. December 1, 1985; 45 (6): 1932-40.


Membrane-related specializations associated with acetylcholine receptor aggregates induced by electric fields., Luther PW., J Cell Biol. January 1, 1985; 100 (1): 235-44.


Acetylcholine receptor aggregation parallels the deposition of a basal lamina proteoglycan during development of the neuromuscular junction., Anderson MJ., J Cell Biol. November 1, 1984; 99 (5): 1769-84.


Aggregates of acetylcholine receptors are associated with plaques of a basal lamina heparan sulfate proteoglycan on the surface of skeletal muscle fibers., Anderson MJ., J Cell Biol. November 1, 1983; 97 (5 Pt 1): 1396-411.

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