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Summary Anatomy Item Literature (132) Expression Attributions Wiki
XB-ANAT-793

Papers associated with endocrine cell (and kcnj11)

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Disease progression and search for monogenic diabetes among children with new onset type 1 diabetes negative for ICA, GAD- and IA-2 Antibodies., Pörksen S., BMC Endocr Disord. September 23, 2010; 10 16.      


Functional analysis of two Kir6.2 (KCNJ11) mutations, K170T and E322K, causing neonatal diabetes., Tarasov AI., Diabetes Obes Metab. November 1, 2007; 9 Suppl 2 46-55.


Iptakalim, a vascular ATP-sensitive potassium (KATP) channel opener, closes rat pancreatic beta-cell KATP channels and increases insulin release., Misaki N., J Pharmacol Exp Ther. August 1, 2007; 322 (2): 871-8.


Scavenging of 14-3-3 proteins reveals their involvement in the cell-surface transport of ATP-sensitive K+ channels., Heusser K., J Cell Sci. October 15, 2006; 119 (Pt 20): 4353-63.


Functional effects of naturally occurring KCNJ11 mutations causing neonatal diabetes on cloned cardiac KATP channels., Tammaro P., J Physiol. February 15, 2006; 571 (Pt 1): 3-14.


The novel diazoxide analog 3-isopropylamino-7-methoxy-4H-1,2,4-benzothiadiazine 1,1-dioxide is a selective Kir6.2/SUR1 channel opener., Dabrowski M., Diabetes. June 1, 2002; 51 (6): 1896-906.


Structural basis for the interference between nicorandil and sulfonylurea action., Reimann F., Diabetes. October 1, 2001; 50 (10): 2253-9.


Glimepiride block of cloned beta-cell, cardiac and smooth muscle K(ATP) channels., Song DK., Br J Pharmacol. May 1, 2001; 133 (1): 193-9.


Effects of mitiglinide (S 21403) on Kir6.2/SUR1, Kir6.2/SUR2A and Kir6.2/SUR2B types of ATP-sensitive potassium channel., Reimann F., Br J Pharmacol. April 1, 2001; 132 (7): 1542-8.


Nucleotide modulation of pinacidil stimulation of the cloned K(ATP) channel Kir6.2/SUR2A., Gribble FM., Mol Pharmacol. June 1, 2000; 57 (6): 1256-61.


Altered functional properties of KATP channel conferred by a novel splice variant of SUR1., Sakura H., J Physiol. December 1, 1999; 521 Pt 2 337-50.


In vitro mechanism of action on insulin release of S-22068, a new putative antidiabetic compound., Le Brigand L., Br J Pharmacol. November 1, 1999; 128 (5): 1021-6.


Interaction of vanadate with the cloned beta cell K(ATP) channel., Proks P., J Biol Chem. September 3, 1999; 274 (36): 25393-7.


Differential sensitivity of beta-cell and extrapancreatic K(ATP) channels to gliclazide., Gribble FM., Diabetologia. July 1, 1999; 42 (7): 845-8.


KATP channel inhibition by ATP requires distinct functional domains of the cytoplasmic C terminus of the pore-forming subunit., Drain P., Proc Natl Acad Sci U S A. November 10, 1998; 95 (23): 13953-8.


Mechanism of cloned ATP-sensitive potassium channel activation by oleoyl-CoA., Gribble FM., J Biol Chem. October 9, 1998; 273 (41): 26383-7.


Molecular analysis of ATP-sensitive K channel gating and implications for channel inhibition by ATP., Trapp S., J Gen Physiol. September 1, 1998; 112 (3): 333-49.                


Mechanism of ATP-sensitive K channel inhibition by sulfhydryl modification., Trapp S., J Gen Physiol. September 1, 1998; 112 (3): 325-32.          


MgATP activates the beta cell KATP channel by interaction with its SUR1 subunit., Gribble FM., Proc Natl Acad Sci U S A. June 9, 1998; 95 (12): 7185-90.


Phentolamine block of KATP channels is mediated by Kir6.2., Proks P., Proc Natl Acad Sci U S A. October 14, 1997; 94 (21): 11716-20.


The interaction of nucleotides with the tolbutamide block of cloned ATP-sensitive K+ channel currents expressed in Xenopus oocytes: a reinterpretation., Gribble FM., J Physiol. October 1, 1997; 504 ( Pt 1) 35-45.


Activation and inhibition of K-ATP currents by guanine nucleotides is mediated by different channel subunits., Trapp S., Proc Natl Acad Sci U S A. August 5, 1997; 94 (16): 8872-7.


Properties of cloned ATP-sensitive K+ currents expressed in Xenopus oocytes., Gribble FM., J Physiol. January 1, 1997; 498 ( Pt 1) 87-98.


Sequence variations in the human Kir6.2 gene, a subunit of the beta-cell ATP-sensitive K-channel: no association with NIDDM in while Caucasian subjects or evidence of abnormal function when expressed in vitro., Sakura H., Diabetologia. October 1, 1996; 39 (10): 1233-6.

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