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Fig. 3. Late embryological defects caused by E2, BPA, and BPAF. A) Control tadpole at 96 hours of development compared to tadpoles exposed to 10 μM estradiol (B), BPA at 10 (C) and 25 μM (D, E), and BPAF at 0.03 μM (F). E2 caused tail flexures, severe pigmentation reduction, long loosely coiled gut, and a ventral blister in 100% of embryos (arrows). BPA caused truncated body axis and body axis defects, bent tails, and head and eye defects (C–E, arrows). BPAF caused shorter tails, craniofacial defects, ventral blisters, edema and peritoneal effusion in over 90% of surviving embryos (F, arrows). Image published in: Arancio AL et al. (2019) © 2019 The Author(s). This image is reproduced with permission of the journal and the copyright holder. This is an open-access article distributed under the terms of the Creative Commons Attribution license Larger Image Printer Friendly View

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