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Summary Expression Phenotypes Gene Literature (14) GO Terms (8) Nucleotides (103) Proteins (60) Interactants (108) Wiki
XB-GENEPAGE-1001814

Papers associated with kcnn1



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Alternative splice isoforms of small conductance calcium-activated SK2 channels differ in molecular interactions and surface levels., Scholl ES, Pirone A, Cox DH, Duncan RK, Jacob MH., Channels (Austin). January 1, 2014; 8 (1): 62-75.                            


Evolution of vertebrate central nervous system is accompanied by novel expression changes of duplicate genes., Chen Y, Chen Y, Ding Y, Zhang Z, Wang W, Chen JY, Ueno N, Mao B., J Genet Genomics. December 20, 2011; 38 (12): 577-84.                                                                                                                                                          


FHL-2 suppresses VEGF-induced phosphatidylinositol 3-kinase/Akt activation via interaction with sphingosine kinase-1., Hayashi H, Nakagami H, Takami Y, Koriyama H, Mori M, Tamai K, Sun J, Nagao K, Morishita R, Kaneda Y., Arterioscler Thromb Vasc Biol. June 1, 2009; 29 (6): 909-14.


5-HT1A receptors modulate small-conductance Ca2+-activated K+ channels., Grunnet M, Jespersen T, Perrier JF., J Neurosci Res. December 15, 2004; 78 (6): 845-54.


Halothane inhibits an intermediate conductance Ca2+-activated K+ channel by acting at the extracellular side of the ionic pore., Hashiguchi-Ikeda M, Namba T, Ishii TM, Hisano T, Fukuda K., Anesthesiology. December 1, 2003; 99 (6): 1340-5.


Small conductance Ca2+-activated K+ channels formed by the expression of rat SK1 and SK2 genes in HEK 293 cells., Benton DC, Monaghan AS, Hosseini R, Bahia PK, Haylett DG, Moss GW., J Physiol. November 15, 2003; 553 (Pt 1): 13-9.


Regional differences in distribution and functional expression of small-conductance Ca2+-activated K+ channels in rat brain., Sailer CA, Hu H, Kaufmann WA, Trieb M, Schwarzer C, Storm JF, Knaus HG., J Neurosci. November 15, 2002; 22 (22): 9698-707.


ATP-dependent activation of the intermediate conductance, Ca2+-activated K+ channel, hIK1, is conferred by a C-terminal domain., Gerlach AC, Syme CA, Giltinan L, Adelman JP, Devors DC., J Biol Chem. June 15, 2001; 276 (24): 10963-70.


Apamin interacts with all subtypes of cloned small-conductance Ca2+-activated K+ channels., Grunnet M, Jensen BS, Olesen SP, Klaerke DA., Pflugers Arch. January 1, 2001; 441 (4): 544-50.


Inhibition of the human intermediate conductance Ca(2+)-activated K(+) channel, hIK1, by volatile anesthetics., Namba T, Ishii TM, Ikeda M, Hisano T, Itoh T, Hirota K, Adelman JP, Fukuda K., Eur J Pharmacol. April 28, 2000; 395 (2): 95-101.


The pharmacology of hSK1 Ca2+-activated K+ channels expressed in mammalian cell lines., Shah M, Haylett DG., Br J Pharmacol. February 1, 2000; 129 (4): 627-30.


Bicuculline block of small-conductance calcium-activated potassium channels., Khawaled R, Bruening-Wright A, Adelman JP, Maylie J., Pflugers Arch. August 1, 1999; 438 (3): 314-21.


Determinants of apamin and d-tubocurarine block in SK potassium channels., Ishii TM, Maylie J, Adelman JP., J Biol Chem. September 12, 1997; 272 (37): 23195-200.


Human and Xenopus cDNAs encoding budding yeast Cdc7-related kinases: in vitro phosphorylation of MCM subunits by a putative human homologue of Cdc7., Sato N, Arai K, Masai H., EMBO J. July 16, 1997; 16 (14): 4340-51.

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