Papers associated with kcnj2

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	Extracellular cysteine disulfide bond break at Cys122 disrupts PIP 2 -dependent Kir2.1 channel function and leads to arrhythmias in Andersen-Tawil Syndrome., Cruz FM, Macías Á, Moreno-Manuel AI, Gutiérrez LK, Vera-Pedrosa ML, Martínez-Carrascoso I, Pérez PS, Ruiz Robles JM, Bermúdez-Jiménez FJ, Díaz-Agustín A, Martínez de Benito F, Santiago SA, Braza-Boils A, Martín-Martínez M, Gutierrez-Rodríguez M, Bernal JA, Zorio E, Jiménez-Jaimez J, Jalife J., bioRxiv. June 8, 2023;
	Expanding the CRISPR/Cas genome-editing scope in Xenopus tropicalis., Shi Z, Jiang H, Liu G, Shi S, Zhang X, Chen Y., Cell Biosci. July 8, 2022; 12 (1): 104.
	Bioelectric signaling: Reprogrammable circuits underlying embryogenesis, regeneration, and cancer., Levin M., Cell. April 15, 2021;
	Identification of a unique endoplasmic retention motif in the Xenopus GIRK5 channel and its contribution to oocyte maturation., Rangel-Garcia CI, Salvador C, Chavez-Garcia K, Diaz-Bello B, Lopez-Gonzalez Z, Vazquez-Cruz L, Angel Vazquez-Martinez J, Ortiz-Navarrete V, Riveros-Rosas H, Escobar LI., FEBS Open Bio. April 1, 2021; 11 (4): 1093-1108.
	Electrophysiological effects of non-vitamin K antagonist oral anticoagulants on atrial repolarizing potassium channels., Wiedmann F, Schlund D, Kraft M, Nietfeld J, Katus HA, Schmidt C, Thomas D., Europace. September 1, 2020; 22 (9): 1409-1418.
	Andersen-Tawil Syndrome Is Associated With Impaired PIP2 Regulation of the Potassium Channel Kir2.1., Handklo-Jamal R, Meisel E, Yakubovich D, Vysochek L, Beinart R, Glikson M, McMullen JR, Dascal N, Nof E, Oz S., Front Pharmacol. April 7, 2020; 11 672.
	Preventing Ethanol-Induced Brain and Eye Morphology Defects Using Optogenetics., Pai VP, Adams DS., Bioelectricity. December 1, 2019; 1 (4): 260-272.
	Nocturnal Atrial Fibrillation Caused by Mutation in KCND2, Encoding Pore-Forming (α) Subunit of the Cardiac Kv4.2 Potassium Channel., Drabkin M, Zilberberg N, Menahem S, Mulla W, Halperin D, Yogev Y, Wormser O, Perez Y, Kadir R, Etzion Y, Katz A, Birk OS., Circ Genom Precis Med. November 1, 2018; 11 (11): e002293.
	Dual Mechanism for Inhibition of Inwardly Rectifying Kir2.x Channels by Quinidine Involving Direct Pore Block and PIP2-interference., Koepple C, Scherer D, Seyler C, Scholz E, Thomas D, Katus HA, Zitron E., J Pharmacol Exp Ther. May 1, 2017; 361 (2): 209-218.
	Bioelectric signalling via potassium channels: a mechanism for craniofacial dysmorphogenesis in KCNJ2-associated Andersen-Tawil Syndrome., Adams DS, Uzel SG, Akagi J, Wlodkowic D, Andreeva V, Yelick PC, Devitt-Lee A, Pare JF, Levin M., J Physiol. June 15, 2016; 594 (12): 3245-70.
	Driving force-dependent block by internal Ba(2+) on the Kir2.1 channel: Mechanistic insight into inward rectification., Hsieh CP, Kuo CC, Huang CW., Biophys Chem. July 1, 2015; 202 40-57.
	Down-regulation of inwardly rectifying Kir2.1 K+ channels by human parvovirus B19 capsid protein VP1., Ahmed M, Elvira B, Almilaji A, Bock CT, Kandolf R, Lang F., J Membr Biol. April 1, 2015; 248 (2): 223-9.
	SPAK and OSR1 Sensitive Kir2.1 K+ Channels., Fezai M, Ahmed M, Hosseinzadeh Z, Elvira B, Lang F., Neurosignals. January 1, 2015; 23 (1): 20-33.
	Class III antiarrhythmic drug dronedarone inhibits cardiac inwardly rectifying Kir2.1 channels through binding at residue E224., Xynogalos P, Seyler C, Scherer D, Koepple C, Scholz EP, Thomas D, Katus HA, Zitron E., Naunyn Schmiedebergs Arch Pharmacol. December 1, 2014; 387 (12): 1153-61.
	Gating of the kir2.1 channel at the bundle crossing region by intracellular spermine and other cations., Huang CW, Kuo CC., J Cell Physiol. November 1, 2014; 229 (11): 1703-21.
	Genetically induced dysfunctions of Kir2.1 channels: implications for short QT3 syndrome and autism-epilepsy phenotype., Ambrosini E, Sicca F, Brignone MS, D'Adamo MC, Napolitano C, Servettini I, Moro F, Ruan Y, Guglielmi L, Pieroni S, Servillo G, Lanciotti A, Valvo G, Catacuzzeno L, Franciolini F, Molinari P, Marchese M, Grottesi A, Guerrini R, Santorelli FM, Priori S, Pessia M., Hum Mol Genet. September 15, 2014; 23 (18): 4875-86.
	A splice variant of the two-pore domain potassium channel TREK-1 with only one pore domain reduces the surface expression of full-length TREK-1 channels., Rinné S, Renigunta V, Schlichthörl G, Zuzarte M, Bittner S, Meuth SG, Decher N, Daut J, Preisig-Müller R., Pflugers Arch. August 1, 2014; 466 (8): 1559-70.
	P639Amiodarone and dronedarone inhibit inwardly rectifying Kir2.1 channels, but not Kir2.2 and Kir2.3 channels., Seyler C, Xynogalos P, Scherer D, Koepple C, Scholz E, Thomas D, Katus H, Zitron E., Cardiovasc Res. July 15, 2014; 103 Suppl 1 S116.
	A Kir3.4 mutation causes Andersen-Tawil syndrome by an inhibitory effect on Kir2.1., Kokunai Y, Nakata T, Furuta M, Sakata S, Kimura H, Aiba T, Yoshinaga M, Osaki Y, Nakamori M, Itoh H, Sato T, Kubota T, Kadota K, Shindo K, Mochizuki H, Shimizu W, Horie M, Okamura Y, Ohno K, Takahashi MP., Neurology. March 25, 2014; 82 (12): 1058-64.
	The bundle crossing region is responsible for the inwardly rectifying internal spermine block of the Kir2.1 channel., Huang CW, Kuo CC., Pflugers Arch. February 1, 2014; 466 (2): 275-93.
	Up-regulation of Kir2.1 (KCNJ2) by the serum & glucocorticoid inducible SGK3., Munoz C, Pakladok T, Almilaji A, Elvira B, Decher N, Shumilina E, Lang F., Cell Physiol Biochem. January 1, 2014; 33 (2): 491-500.
	Lack of negatively charged residues at the external mouth of Kir2.2 channels enable the voltage-dependent block by external Mg2+., Li J, Xie X, Liu J, Yu H, Zhang S, Zhan Y, Zhang H, Logothetis DE, An H., PLoS One. January 1, 2014; 9 (10): e111372.
	Non dominant-negative KCNJ2 gene mutations leading to Andersen-Tawil syndrome with an isolated cardiac phenotype., Limberg MM, Zumhagen S, Netter MF, Coffey AJ, Grace A, Rogers J, Böckelmann D, Rinné S, Stallmeyer B, Decher N, Schulze-Bahr E., Basic Res Cardiol. May 1, 2013; 108 (3): 353.
	Up-regulation of the inwardly rectifying K⁺ channel Kir2.1 (KCNJ2) by protein kinase B (PKB/Akt) and PIKfyve., Munoz C, Almilaji A, Setiawan I, Föller M, Lang F., J Membr Biol. March 1, 2013; 246 (3): 189-97.
	Voltage-dependent inhibition of outward Kir2.1 currents by extracellular spermine., Chang HK, Shieh RC., Biochim Biophys Acta. February 1, 2013; 1828 (2): 765-75.
	Conformational changes underlying pore dilation in the cytoplasmic domain of mammalian inward rectifier K+ channels., Inanobe A, Nakagawa A, Kurachi Y., PLoS One. January 1, 2013; 8 (11): e79844.
	Interactions of external K+ and internal blockers in a weak inward-rectifier K+ channel., Yang L, Edvinsson J, Palmer LG., J Gen Physiol. November 1, 2012; 140 (5): 529-40.
	Inhibition of cardiac Kir2.1-2.3 channels by beta3 adrenoreceptor antagonist SR 59230A., Kulzer M, Seyler C, Welke F, Scherer D, Xynogalos P, Scholz EP, Thomas D, Becker R, Karle CA, Katus HA, Zitron E., Biochem Biophys Res Commun. July 27, 2012; 424 (2): 315-20.
	Revisiting inward rectification: K ions permeate through Kir2.1 channels during high-affinity block by spermidine., Liu TA, Chang HK, Shieh RC., J Gen Physiol. March 1, 2012; 139 (3): 245-59.
	Discovery, characterization, and structure-activity relationships of an inhibitor of inward rectifier potassium (Kir) channels with preference for Kir2.3, Kir3.x, and Kir7.1., Raphemot R, Lonergan DF, Nguyen TT, Utley T, Lewis LM, Kadakia R, Weaver CD, Gogliotti R, Hopkins C, Lindsley CW, Denton JS., Front Pharmacol. November 30, 2011; 2 75.
	Extracellular K+ elevates outward currents through Kir2.1 channels by increasing single-channel conductance., Liu TA, Chang HK, Shieh RC., Biochim Biophys Acta. June 1, 2011; 1808 (6): 1772-8.
	Inhibition of Kir2.1 (KCNJ2) by the AMP-activated protein kinase., Alesutan I, Munoz C, Sopjani M, Dërmaku-Sopjani M, Michael D, Fraser S, Kemp BE, Seebohm G, Föller M, Lang F., Biochem Biophys Res Commun. May 20, 2011; 408 (4): 505-10.
	Inhibition of g protein-activated inwardly rectifying k channels by phencyclidine., Kobayashi T, Nishizawa D, Ikeda K., Curr Neuropharmacol. March 1, 2011; 9 (1): 244-6.
	Cholesterol sensitivity of KIR2.1 is controlled by a belt of residues around the cytosolic pore., Rosenhouse-Dantsker A, Logothetis DE, Levitan I., Biophys J. January 19, 2011; 100 (2): 381-9.
	Inhibition of G protein-activated inwardly rectifying K+ channels by different classes of antidepressants., Kobayashi T, Washiyama K, Ikeda K., PLoS One. January 1, 2011; 6 (12): e28208.
	Comparative analysis of cholesterol sensitivity of Kir channels: role of the CD loop., Rosenhouse-Dantsker A, Leal-Pinto E, Logothetis DE, Levitan I., Channels (Austin). January 1, 2010; 4 (1): 63-6.
	Selection of inhibitor-resistant viral potassium channels identifies a selectivity filter site that affects barium and amantadine block., Chatelain FC, Gazzarrini S, Fujiwara Y, Arrigoni C, Domigan C, Ferrara G, Pantoja C, Thiel G, Moroni A, Minor DL., PLoS One. October 16, 2009; 4 (10): e7496.
	KCNE1 and KCNE3 beta-subunits regulate membrane surface expression of Kv12.2 K(+) channels in vitro and form a tripartite complex in vivo., Clancy SM, Chen B, Bertaso F, Mamet J, Jegla T., PLoS One. July 22, 2009; 4 (7): e6330.
	Pregnenolone sulfate potentiates the inwardly rectifying K channel Kir2.3., Kobayashi T, Washiyama K, Ikeda K., PLoS One. July 21, 2009; 4 (7): e6311.
	Heteromeric assembly of inward rectifier channel subunit Kir2.1 with Kir3.1 and with Kir3.4., Ishihara K, Yamamoto T, Kubo Y., Biochem Biophys Res Commun. March 20, 2009; 380 (4): 832-7.
	Structural changes in the cytoplasmic pore of the Kir1.1 channel during pHi-gating probed by FRET., Lee JR, Shieh RC., J Biomed Sci. March 6, 2009; 16 29.
	K+ binding in the G-loop and water cavity facilitates Ba2+ movement in the Kir2.1 channel., Chang HK, Marton LJ, Liang KK, Shieh RC., Biochim Biophys Acta. February 1, 2009; 1788 (2): 500-6.
	Phosphatidylinositol-4,5-bisphosphate (PIP2) regulation of strong inward rectifier Kir2.1 channels: multilevel positive cooperativity., Xie LH, John SA, Ribalet B, Weiss JN., J Physiol. April 1, 2008; 586 (7): 1833-48.
	Kir2.x inward rectifier potassium channels are differentially regulated by adrenergic alpha1A receptors., Zitron E, Günth M, Scherer D, Kiesecker C, Kulzer M, Bloehs R, Scholz EP, Thomas D, Weidenhammer C, Kathöfer S, Bauer A, Katus HA, Karle CA., J Mol Cell Cardiol. January 1, 2008; 44 (1): 84-94.
	Differential effects of volatile and intravenous anesthetics on the activity of human TASK-1., Putzke C, Hanley PJ, Schlichthörl G, Preisig-Müller R, Rinné S, Anetseder M, Eckenhoff R, Berkowitz C, Vassiliou T, Wulf H, Eberhart L., Am J Physiol Cell Physiol. October 1, 2007; 293 (4): C1319-26.
	Depolarization activates the phosphoinositide phosphatase Ci-VSP, as detected in Xenopus oocytes coexpressing sensors of PIP2., Murata Y, Okamura Y., J Physiol. September 15, 2007; 583 (Pt 3): 875-89.
	Impaired interaction between the slide helix and the C-terminus of Kir2.1: a novel mechanism of Andersen syndrome., Decher N, Renigunta V, Zuzarte M, Soom M, Heinemann SH, Timothy KW, Keating MT, Daut J, Sanguinetti MC, Splawski I., Cardiovasc Res. September 1, 2007; 75 (4): 748-57.
	H bonding at the helix-bundle crossing controls gating in Kir potassium channels., Rapedius M, Fowler PW, Shang L, Sansom MS, Tucker SJ, Baukrowitz T., Neuron. August 16, 2007; 55 (4): 602-14.
	A di-acidic sequence motif enhances the surface expression of the potassium channel TASK-3., Zuzarte M, Rinné S, Schlichthörl G, Schubert A, Daut J, Preisig-Müller R., Traffic. August 1, 2007; 8 (8): 1093-100.
	Activation of inwardly rectifying Kir2.x potassium channels by beta 3-adrenoceptors is mediated via different signaling pathways with a predominant role of PKC for Kir2.1 and of PKA for Kir2.2., Scherer D, Kiesecker C, Kulzer M, Günth M, Scholz EP, Kathöfer S, Thomas D, Maurer M, Kreuzer J, Bauer A, Katus HA, Karle CA, Zitron E., Naunyn Schmiedebergs Arch Pharmacol. July 1, 2007; 375 (5): 311-22.

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