Click here to close Hello! We notice that you are using Internet Explorer, which is not supported by Xenbase and may cause the site to display incorrectly. We suggest using a current version of Chrome, FireFox, or Safari.

Summary Expression Phenotypes Gene Literature (9) GO Terms (2) Nucleotides (126) Proteins (50) Interactants (41) Wiki
XB-GENEPAGE-5824174

Papers associated with mtss1.2



???displayGene.coCitedPapers???

???pagination.result.count???

???pagination.result.page??? 1

Sort Newest To Oldest Sort Oldest To Newest

Sequence and timing of early cranial skeletal development in Xenopus laevis., Lukas P, Olsson L., J Morphol. January 1, 2018; 279 (1): 62-74.            

Involvement of Slit-Robo signaling in the development of the posterior commissure and concomitant swimming behavior in Xenopus laevis., Tosa Y, Tsukano K, Itoyama T, Fukagawa M, Nii Y, Ishikawa R, Suzuki KT, Fukui M, Kawaguchi M, Murakami Y., Zoological Lett. June 15, 2015; 1 28.                      

Novel animal pole-enriched maternal mRNAs are preferentially expressed in neural ectoderm., Grant PA, Yan B, Johnson MA, Johnson DL, Moody SA., Dev Dyn. March 1, 2014; 243 (3): 478-96.                                        

MIM regulates vertebrate neural tube closure., Liu W, Komiya Y, Mezzacappa C, Khadka DK, Runnels L, Habas R., Development. May 1, 2011; 138 (10): 2035-47.                            

Functional analysis of novel mutations in y(+)LAT-1 amino acid transporter gene causing lysinuric protein intolerance (LPI)., Mykkänen J, Torrents D, Pineda M, Camps M, Yoldi ME, Horelli-Kuitunen N, Huoponen K, Heinonen M, Oksanen J, Simell O, Savontaus ML, Zorzano A, Palacín M, Aula P., Hum Mol Genet. February 12, 2000; 9 (3): 431-8.

Boy with syndactylies, macrocephaly, and severe skeletal dysplasia: not a new syndrome, but two dominant mutations (GLI3 E543X and COL2A1 G973R) in the same individual., Sobetzko D, Eich G, Kalff-Suske M, Grzeschik KH, Superti-Furga A., Am J Med Genet. January 31, 2000; 90 (3): 239-42.

Point mutations throughout the GLI3 gene cause Greig cephalopolysyndactyly syndrome., Kalff-Suske M, Wild A, Topp J, Wessling M, Jacobsen EM, Bornholdt D, Engel H, Heuer H, Aalfs CM, Ausems MG, Barone R, Herzog A, Heutink P, Homfray T, Gillessen-Kaesbach G, König R, Kunze J, Meinecke P, Müller D, Rizzo R, Strenge S, Superti-Furga A, Grzeschik KH., Hum Mol Genet. September 1, 1999; 8 (9): 1769-77.

Point mutations in human GLI3 cause Greig syndrome., Wild A, Kalff-Suske M, Vortkamp A, Bornholdt D, König R, Grzeschik KH., Hum Mol Genet. October 1, 1997; 6 (11): 1979-84.

Developmental potential., Holmgren RA, Engel JD., Genes Dev. February 1, 1992; 6 (2): 161-5.

???pagination.result.page??? 1