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XB-GENEPAGE-6032634
Papers associated with kcnc4
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Kv3.1 channelopathy: a novel loss-of-function variant and the mechanistic basis of its clinical phenotypes., Li X, Zheng Y, Li S, Nair U, Sun C, Zhao C, Lu J, Zhang VW, Maljevic S, Petrou S, Lin J., Ann Transl Med. September 1, 2021; 9 (18): 1397. |
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KCNC1-related disorders: new de novo variants expand the phenotypic spectrum., Park J, Koko M, Hedrich UBS, Hermann A, Cremer K, Haberlandt E, Grimmel M, Alhaddad B, Beck-Woedl S, Harrer M, Karall D, Kingelhoefer L, Tzschach A, Matthies LC, Strom TM, Ringelstein EB, Sturm M, Engels H, Wolff M, Lerche H, Haack TB., Ann Clin Transl Neurol. July 1, 2019; 6 (7): 1319-1326.  |
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Modeling-independent elucidation of inactivation pathways in recombinant and native A-type Kv channels., Fineberg JD, Ritter DM, Covarrubias M., J Gen Physiol. November 1, 2012; 140 (5): 513-27.  |
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1,4-Diazabicyclo[2.2.2]octane derivatives: a novel class of voltage-gated potassium channel blockers., Gordon E, Cohen JL, Engel R, Abbott GW., Mol Pharmacol. March 1, 2006; 69 (3): 718-26. |
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A role for frequenin, a Ca2+-binding protein, as a regulator of Kv4 K+-currents., Nakamura TY, Pountney DJ, Ozaita A, Nandi S, Ueda S, Rudy B, Coetzee WA., Proc Natl Acad Sci U S A. October 23, 2001; 98 (22): 12808-13. |
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MiRP2 forms potassium channels in skeletal muscle with Kv3.4 and is associated with periodic paralysis., Abbott GW, Butler MH, Bendahhou S, Dalakas MC, Ptacek LJ, Goldstein SA., Cell. January 26, 2001; 104 (2): 217-31. |
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NMR structure and functional characteristics of the hydrophilic N terminus of the potassium channel beta-subunit Kvbeta1.1., Wissmann R, Baukrowitz T, Kalbacher H, Kalbitzer HR, Ruppersberg JP, Pongs O, Antz C, Fakler B., J Biol Chem. December 10, 1999; 274 (50): 35521-5. |
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Interactions between multiple phosphorylation sites in the inactivation particle of a K+ channel. Insights into the molecular mechanism of protein kinase C action., Beck EJ, Sorensen RG, Slater SJ, Covarrubias M., J Gen Physiol. July 1, 1998; 112 (1): 71-84.  |
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Sea anemone peptides with a specific blocking activity against the fast inactivating potassium channel Kv3.4., Diochot S, Schweitz H, Béress L, Lazdunski M., J Biol Chem. March 20, 1998; 273 (12): 6744-9. |
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Modes of regulation of shab K+ channel activity by the Kv8.1 subunit., Salinas M, de Weille J, Guillemare E, Lazdunski M, Hugnot JP., J Biol Chem. March 28, 1997; 272 (13): 8774-80. |
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Functional characterization of Kv channel beta-subunits from rat brain., Heinemann SH, Rettig J, Graack HR, Pongs O., J Physiol. June 15, 1996; 493 ( Pt 3) 625-33. |
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Susceptibility of cloned K+ channels to reactive oxygen species., Duprat F, Guillemare E, Romey G, Fink M, Lesage F, Lazdunski M, Honore E., Proc Natl Acad Sci U S A. December 5, 1995; 92 (25): 11796-800. |
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Alcohols inhibit a cloned potassium channel at a discrete saturable site. Insights into the molecular basis of general anesthesia., Covarrubias M, Vyas TB, Escobar L, Wei A., J Biol Chem. August 18, 1995; 270 (33): 19408-16. |
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Modulation of K+ channels by hydrogen peroxide., Vega-Saenz de Miera E, Rudy B., Biochem Biophys Res Commun. August 14, 1992; 186 (3): 1681-7. |
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Cloning of ShIII (Shaw-like) cDNAs encoding a novel high-voltage-activating, TEA-sensitive, type-A K+ channel., Vega-Saenz de Miera E, Moreno H, Fruhling D, Kentros C, Rudy B., Proc Biol Sci. April 22, 1992; 248 (1321): 9-18. |
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Cloning of a human cDNA expressing a high voltage-activating, TEA-sensitive, type-A K+ channel which maps to chromosome 1 band p21., Rudy B, Sen K, Vega-Saenz de Miera E, Lau D, Ried T, Ward DC., J Neurosci Res. July 1, 1991; 29 (3): 401-12. |
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