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Summary Expression Gene Literature (113) GO Terms (9) Nucleotides (27) Proteins (13) Interactants (100) Wiki
XB-GENEPAGE-6084576

Papers associated with kcnj11

Search for kcnj11 morpholinos using Textpresso

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1 paper(s) referencing morpholinos

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Permanent neonatal diabetes: combining sulfonylureas with insulin may be an effective treatment., Misra S, Vedovato N, Cliff E, De Franco E, Hattersley AT, Ashcroft FM, Oliver NS., Diabet Med. June 13, 2018;


Xenopus as a model system for studying pancreatic development and diabetes., Kofent J, Spagnoli FM., Semin Cell Dev Biol. March 1, 2016; 51 106-16.  


Sensitivity of KATP channels to cellular metabolic disorders and the underlying structural basis., Li CG, Cui WY, Wang H., Acta Pharmacol Sin. January 1, 2016; 37 (1): 134-42.        


Bioelectric signalling via potassium channels: a mechanism for craniofacial dysmorphogenesis in KCNJ2-associated Andersen-Tawil Syndrome., Adams DS, Uzel SG, Akagi J, Wlodkowic D, Andreeva V, Yelick PC, Devitt-Lee A, Pare JF, Levin M., J Physiol. January 1, 2016; 594 (12): 3245-70.                              


Successful transfer to sulfonylureas in KCNJ11 neonatal diabetes is determined by the mutation and duration of diabetes., Babiker T, Vedovato N, Patel K, Thomas N, Finn R, Männikkö R, Chakera AJ, Flanagan SE, Shepherd MH, Ellard S, Ashcroft FM, Hattersley AT., Diabetologia. January 1, 2016; 59 (6): 1162-6.    


Neonatal diabetes caused by a homozygous KCNJ11 mutation demonstrates that tiny changes in ATP sensitivity markedly affect diabetes risk., Vedovato N, Cliff E, Proks P, Poovazhagi V, Flanagan SE, Ellard S, Hattersley AT, Ashcroft FM., Diabetologia. January 1, 2016; 59 (7): 1430-1436.        


Kir6.2 activation by sulfonylurea receptors: a different mechanism of action for SUR1 and SUR2A subunits via the same residues., Principalli MA, Dupuis JP, Moreau CJ, Vivaudou M, Revilloud J., Physiol Rep. September 1, 2015; 3 (9):         


Distinct action of the α-glucosidase inhibitor miglitol on SGLT3, enteroendocrine cells, and GLP1 secretion., Lee EY, Kaneko S, Jutabha P, Zhang X, Seino S, Jomori T, Anzai N, Miki T., J Endocrinol. March 1, 2015; 224 (3): 205-14.            


Sulfonylureas suppress the stimulatory action of Mg-nucleotides on Kir6.2/SUR1 but not Kir6.2/SUR2A KATP channels: a mechanistic study., Proks P, de Wet H, Ashcroft FM., J Gen Physiol. November 1, 2014; 144 (5): 469-86.                  


Recessive mutations in PCBD1 cause a new type of early-onset diabetes., Simaite D, Kofent J, Gong M, Rüschendorf F, Jia S, Arn P, Bentler K, Ellaway C, Kühnen P, Hoffmann GF, Blau N, Spagnoli FM, Hübner N, Raile K., Diabetes. October 1, 2014; 63 (10): 3557-64.


Intracellular ATP does not inhibit Slo2.1 K+ channels., Garg P, Sanguinetti MC., Physiol Rep. September 1, 2014; 2 (9):         


The unusual stoichiometry of ADP activation of the KATP channel., Hosy E, Vivaudou M., Front Physiol. January 1, 2014; 5 11.        


Molecular mechanism of sulphonylurea block of K(ATP) channels carrying mutations that impair ATP inhibition and cause neonatal diabetes., Proks P, de Wet H, Ashcroft FM., Diabetes. November 1, 2013; 62 (11): 3909-19.              


A universally conserved residue in the SUR1 subunit of the KATP channel is essential for translating nucleotide binding at SUR1 into channel opening., de Wet H, Shimomura K, Aittoniemi J, Ahmad N, Lafond M, Sansom MS, Ashcroft FM., J Physiol. October 15, 2012; 590 (20): 5025-36.            


Engineering of an artificial light-modulated potassium channel., Caro LN, Moreau CJ, Estrada-Mondragón A, Ernst OP, Vivaudou M., PLoS One. January 1, 2012; 7 (8): e43766.        


Forced gating motions by a substituted titratable side chain at the bundle crossing of a potassium channel., Khurana A, Shao ES, Kim RY, Vilin YY, Huang X, Yang R, Kurata HT., J Biol Chem. October 21, 2011; 286 (42): 36686-93.


A conserved tryptophan at the membrane-water interface acts as a gatekeeper for Kir6.2/SUR1 channels and causes neonatal diabetes when mutated., Männikkö R, Stansfeld PJ, Ashcroft AS, Hattersley AT, Sansom MS, Ellard S, Ashcroft FM., J Physiol. July 1, 2011; 589 (Pt 13): 3071-83.


Mutations of the same conserved glutamate residue in NBD2 of the sulfonylurea receptor 1 subunit of the KATP channel can result in either hyperinsulinism or neonatal diabetes., Männikkö R, Flanagan SE, Sim X, Segal D, Hussain K, Ellard S, Hattersley AT, Ashcroft FM., Diabetes. June 1, 2011; 60 (6): 1813-22.              


β2-Adrenergic ion-channel coupled receptors as conformational motion detectors., Caro LN, Moreau CJ, Revilloud J, Vivaudou M., PLoS One. March 2, 2011; 6 (3): e18226.            


The ATP-sensitive K(+)-channel (K(ATP)) controls early left-right patterning in Xenopus and chick embryos., Aw S, Koster JC, Pearson W, Nichols CG, Shi NQ, Carneiro K, Levin M., Dev Biol. October 1, 2010; 346 (1): 39-53.        

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