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Summary Expression Phenotypes Gene Literature (18) GO Terms (5) Nucleotides (65) Proteins (40) Interactants (199) Wiki
XB--1221136

Papers associated with invs



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Modeling congenital kidney diseases in Xenopus laevis., Blackburn ATM, Miller RK., Dis Model Mech. April 9, 2019; 12 (4):       

The nucleoside-diphosphate kinase NME3 associates with nephronophthisis proteins and is required for ciliary function during renal development., Hoff S, Epting D, Falk N, Schroda S, Braun DA, Halbritter J, Hildebrandt F, Kramer-Zucker A, Bergmann C, Walz G, Lienkamp SS., J Biol Chem. September 28, 2018; 293 (39): 15243-15255.            

The ciliopathy-associated CPLANE proteins direct basal body recruitment of intraflagellar transport machinery., Toriyama M, Lee C, Taylor SP, Duran I, Cohn DH, Bruel AL, Tabler JM, Drew K, Kelly MR, Kim S, Park TJ, Braun DA, Pierquin G, Biver A, Wagner K, Malfroot A, Panigrahi I, Franco B, Al-Lami HA, Yeung Y, Choi YJ, University of Washington Center for Mendelian Genomics, Duffourd Y, Faivre L, Rivière JB, Chen J, Liu KJ, Liu KJ, Marcotte EM, Hildebrandt F, Thauvin-Robinet C, Krakow D, Jackson PK, Wallingford JB., Nat Genet. June 1, 2016; 48 (6): 648-56.                              

Using Xenopus to study genetic kidney diseases., Lienkamp SS., Semin Cell Dev Biol. March 1, 2016; 51 117-24.    

Control of vertebrate core planar cell polarity protein localization and dynamics by Prickle 2., Butler MT, Wallingford JB., Development. October 1, 2015; 142 (19): 3429-39.

The Wnt/JNK signaling target gene alcam is required for embryonic kidney development., Cizelsky W, Tata A, Kühl M, Kühl SJ., Development. May 1, 2014; 141 (10): 2064-74.          

ANKS6 is a central component of a nephronophthisis module linking NEK8 to INVS and NPHP3., Hoff S, Halbritter J, Epting D, Frank V, Nguyen TM, van Reeuwijk J, Boehlke C, Schell C, Yasunaga T, Helmstädter M, Mergen M, Filhol E, Boldt K, Horn N, Ueffing M, Otto EA, Eisenberger T, Elting MW, van Wijk JA, Bockenhauer D, Sebire NJ, Rittig S, Vyberg M, Ring T, Pohl M, Pape L, Neuhaus TJ, Elshakhs NA, Koon SJ, Harris PC, Grahammer F, Huber TB, Kuehn EW, Kramer-Zucker A, Bolz HJ, Roepman R, Saunier S, Walz G, Hildebrandt F, Bergmann C, Lienkamp SS., Nat Genet. August 1, 2013; 45 (8): 951-6.                                

Functional aspects of primary cilia in signaling, cell cycle and tumorigenesis., Basten SG, Giles RH., Cilia. April 29, 2013; 2 (1): 6.          

Pronephric tubulogenesis requires Daam1-mediated planar cell polarity signaling., Miller RK, Canny SG, Jang CW, Cho K, Ji H, Wagner DS, Jones EA, Habas R, McCrea PD., J Am Soc Nephrol. September 1, 2011; 22 (9): 1654-64.

Inversin relays Frizzled-8 signals to promote proximal pronephros development., Lienkamp S, Ganner A, Boehlke C, Schmidt T, Arnold SJ, Schäfer T, Romaker D, Schuler J, Hoff S, Powelske C, Eifler A, Krönig C, Bullerkotte A, Nitschke R, Kuehn EW, Kim E, Burkhardt H, Brox T, Ronneberger O, Gloy J, Walz G., Proc Natl Acad Sci U S A. November 23, 2010; 107 (47): 20388-93.                          

Loss of nephrocystin-3 function can cause embryonic lethality, Meckel-Gruber-like syndrome, situs inversus, and renal-hepatic-pancreatic dysplasia., Bergmann C, Fliegauf M, Brüchle NO, Frank V, Olbrich H, Kirschner J, Schermer B, Schmedding I, Kispert A, Kränzlin B, Nürnberg G, Becker C, Grimm T, Girschick G, Lynch SA, Kelehan P, Senderek J, Neuhaus TJ, Stallmach T, Zentgraf H, Nürnberg P, Gretz N, Lo C, Lienkamp S, Schäfer T, Walz G, Benzing T, Zerres K, Omran H., Am J Hum Genet. April 1, 2008; 82 (4): 959-70.

Cilia-driven leftward flow determines laterality in Xenopus., Schweickert A, Weber T, Beyer T, Vick P, Bogusch S, Feistel K, Blum M., Curr Biol. January 9, 2007; 17 (1): 60-6.        

Isolation and characterization of Xenopus laevis homologs of the mouse inv gene and functional analysis of the conserved calmodulin binding sites., Yasuhiko Y, Shiokawa K, Mochizuki T, Asashima M, Yokoyama T., Cell Res. April 1, 2006; 16 (4): 337-46.

Localization and loss-of-function implicates ciliary proteins in early, cytoplasmic roles in left-right asymmetry., Qiu D, Cheng SM, Wozniak L, McSweeney M, Perrone E, Levin M., Dev Dyn. September 1, 2005; 234 (1): 176-89.      

Inversin, the gene product mutated in nephronophthisis type II, functions as a molecular switch between Wnt signaling pathways., Simons M, Gloy J, Ganner A, Bullerkotte A, Bashkurov M, Krönig C, Schermer B, Benzing T, Cabello OA, Jenny A, Mlodzik M, Polok B, Driever W, Obara T, Walz G., Nat Genet. May 1, 2005; 37 (5): 537-43.

The left-right determinant Inversin is a component of node monocilia and other 9+0 cilia., Watanabe D, Saijoh Y, Nonaka S, Sasaki G, Ikawa Y, Yokoyama T, Hamada H., Development. May 1, 2003; 130 (9): 1725-34.

Inversin forms a complex with catenins and N-cadherin in polarized epithelial cells., Nürnberger J, Bacallao RL, Phillips CL., Mol Biol Cell. September 1, 2002; 13 (9): 3096-106.

The left-right determinant inversin has highly conserved ankyrin repeat and IQ domains and interacts with calmodulin., Morgan D, Goodship J, Essner JJ, Vogan KJ, Turnpenny L, Yost HJ, Tabin CJ, Strachan T., Hum Genet. April 1, 2002; 110 (4): 377-84.

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