|
XB-ANAT-3713
Papers associated with left (and kmt2d)
???pagination.result.count??????pagination.result.page??? 1
| Sort Newest To Oldest | Sort Oldest To Newest |
A convergent molecular network underlying autism and congenital heart disease., Rosenthal SB., Cell Syst. November 17, 2021; 12 (11): 1094-1107.e6.  |
|
The histone methyltransferase KMT2D, mutated in Kabuki syndrome patients, is required for neural crest cell formation and migration., Schwenty-Lara J., Hum Mol Genet. January 15, 2020; 29 (2): 305-319.  |
|
Loss of function of Kmt2d, a gene mutated in Kabuki syndrome, affects heart development in Xenopus laevis., Schwenty-Lara J., Dev Dyn. June 1, 2019; 248 (6): 465-476.  |
|
A phospho-dependent mechanism involving NCoR and KMT2D controls a permissive chromatin state at Notch target genes., Oswald F., Nucleic Acids Res. June 2, 2016; 44 (10): 4703-20.  |
|
WDR5 associates with histone H3 methylated at K4 and is essential for H3 K4 methylation and vertebrate development., Wysocka J., Cell. June 17, 2005; 121 (6): 859-72.  |
???pagination.result.page??? 1