Papers associated with left (and kcne1)

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	Pharmacological rescue of specific long QT variants of KCNQ1/KCNE1 channels., Zou X., Front Physiol. January 1, 2022; 13 902224.
	Compound Heterozygous KCNQ1 Mutations Causing Recessive Romano-Ward Syndrome: Functional Characterization by Mutant Co-expression., González-Garrido A., Front Cardiovasc Med. February 22, 2021; 8 625449.
	Polyunsaturated fatty acid analogues differentially affect cardiac NaV, CaV, and KV channels through unique mechanisms., Bohannon BM., Elife. March 24, 2020; 9
	Structure and physiological function of the human KCNQ1 channel voltage sensor intermediate state., Taylor KC., Elife. February 25, 2020; 9
	Deconstruction of an African folk medicine uncovers a novel molecular strategy for therapeutic potassium channel activation., De Silva AM., Sci Adv. November 14, 2018; 4 (11): eaav0824.
	Direct neurotransmitter activation of voltage-gated potassium channels., Manville RW., Nat Commun. May 10, 2018; 9 (1): 1847.
	High incidence of functional ion-channel abnormalities in a consecutive Long QT cohort with novel missense genetic variants of unknown significance., Steffensen AB., Sci Rep. January 12, 2015; 5 10009.
	Impaired ion channel function related to a common KCNQ1 mutation - implications for risk stratification in long QT syndrome 1., Aidery P., Gene. December 10, 2012; 511 (1): 26-33.
	Strong activation of ether-à-go-go-related gene 1 K+ channel isoforms by NS1643 in human embryonic kidney 293 and Chinese hamster ovary cells., Schuster AM., Mol Pharmacol. November 1, 2011; 80 (5): 930-42.
	KCNQ1 subdomains involved in KCNE modulation revealed by an invertebrate KCNQ1 orthologue., Nakajo K., J Gen Physiol. November 1, 2011; 138 (5): 521-35.
	Transmembrane potential of GlyCl-expressing instructor cells induces a neoplastic-like conversion of melanocytes via a serotonergic pathway., Blackiston D., Dis Model Mech. January 1, 2011; 4 (1): 67-85.
	State-dependent electrostatic interactions of S4 arginines with E1 in S2 during Kv7.1 activation., Wu D., J Gen Physiol. June 1, 2010; 135 (6): 595-606.
	KCNE1 and KCNE3 beta-subunits regulate membrane surface expression of Kv12.2 K(+) channels in vitro and form a tripartite complex in vivo., Clancy SM., PLoS One. July 22, 2009; 4 (7): e6330.
	Discovery of a novel activator of KCNQ1-KCNE1 K channel complexes., Mruk K., PLoS One. January 1, 2009; 4 (1): e4236.
	Modulation of potassium channel function confers a hyperproliferative invasive phenotype on embryonic stem cells., Morokuma J., Proc Natl Acad Sci U S A. October 28, 2008; 105 (43): 16608-13.
	KCNQ1 and KCNE1 in the IKs channel complex make state-dependent contacts in their extracellular domains., Xu X., J Gen Physiol. June 1, 2008; 131 (6): 589-603.
	KCNE peptides differently affect voltage sensor equilibrium and equilibration rates in KCNQ1 K+ channels., Rocheleau JM., J Gen Physiol. January 1, 2008; 131 (1): 59-68.
	KCNQ1 and KCNE1 K+ channel components are involved in early left-right patterning in Xenopus laevis embryos., Morokuma J., Cell Physiol Biochem. January 1, 2008; 21 (5-6): 357-72.
	KCNE1 and KCNE3 stabilize and/or slow voltage sensing S4 segment of KCNQ1 channel., Nakajo K., J Gen Physiol. September 1, 2007; 130 (3): 269-81.
	The role of S4 charges in voltage-dependent and voltage-independent KCNQ1 potassium channel complexes., Panaghie G., J Gen Physiol. February 1, 2007; 129 (2): 121-33.
	Secondary structure of a KCNE cytoplasmic domain., Rocheleau JM., J Gen Physiol. December 1, 2006; 128 (6): 721-9.
	KCNE3 truncation mutants reveal a bipartite modulation of KCNQ1 K+ channels., Gage SD., J Gen Physiol. December 1, 2004; 124 (6): 759-71.
	Short- and long-term effects of amiodarone on the two components of cardiac delayed rectifier K(+) current., Kamiya K., Circulation. March 6, 2001; 103 (9): 1317-24.
	Inhibition of IKs channels by HMR 1556., Gögelein H., Naunyn Schmiedebergs Arch Pharmacol. December 1, 2000; 362 (6): 480-8.
	MinK subdomains that mediate modulation of and association with KvLQT1., Tapper AR., J Gen Physiol. September 1, 2000; 116 (3): 379-90.

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