Papers associated with anatomical direction (and cav2)

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	Association of A Novel Splice Site Mutation in P/Q-Type Calcium Channels with Childhood Epilepsy and Late-Onset Slowly Progressive Non-Episodic Cerebellar Ataxia., Stendel C., Int J Mol Sci. May 27, 2020; 21 (11):
	Sigma-1 Receptor Plays a Negative Modulation on N-type Calcium Channel., Zhang K., Front Pharmacol. May 26, 2017; 8 302.
	Honeybee locomotion is impaired by Am-CaV3 low voltage-activated Ca2+ channel antagonist., Rousset M., Sci Rep. February 1, 2017; 7 41782.
	Selective pressure modulation of synaptic voltage-dependent calcium channels-involvement in HPNS mechanism., Aviner B., J Cell Mol Med. October 1, 2016; 20 (10): 1872-88.
	Less is More: Design of a Highly Stable Disulfide-Deleted Mutant of Analgesic Cyclic α-Conotoxin Vc1.1., Yu R., Sci Rep. August 20, 2015; 5 13264.
	A Single Amino Acid Deletion (ΔF1502) in the S6 Segment of CaV2.1 Domain III Associated with Congenital Ataxia Increases Channel Activity and Promotes Ca2+ Influx., Bahamonde MI., PLoS One. January 1, 2015; 10 (12): e0146035.
	The role of voltage-gated calcium channels in neurotransmitter phenotype specification: Coexpression and functional analysis in Xenopus laevis., Lewis BB., J Comp Neurol. August 1, 2014; 522 (11): 2518-31.
	The inhibition of functional expression of calcium channels by prion protein demonstrates competition with α2δ for GPI-anchoring pathways., Alvarez-Laviada A., Biochem J. March 1, 2014; 458 (2): 365-74.
	A post-burst after depolarization is mediated by group i metabotropic glutamate receptor-dependent upregulation of Ca(v)2.3 R-type calcium channels in CA1 pyramidal neurons., Park JY., PLoS Biol. November 16, 2010; 8 (11): e1000534.
	N terminus is key to the dominant negative suppression of Ca(V)2 calcium channels: implications for episodic ataxia type 2., Page KM., J Biol Chem. January 8, 2010; 285 (2): 835-44.
	Cloning and characterization of voltage-gated calcium channel alpha1 subunits in Xenopus laevis during development., Lewis BB., Dev Dyn. November 1, 2009; 238 (11): 2891-902.
	Disruption of the IS6-AID linker affects voltage-gated calcium channel inactivation and facilitation., Findeisen F., J Gen Physiol. March 1, 2009; 133 (3): 327-43.
	The separation of antagonist from agonist effects of trisubstituted purines on CaV2.2 (N-type) channels., Buraei Z., J Neurochem. May 1, 2008; 105 (4): 1450-61.
	Neurite outgrowth and in vivo sensory innervation mediated by a Ca(V)2.2-laminin beta 2 stop signal., Sann SB., J Neurosci. March 5, 2008; 28 (10): 2366-74.
	Depolarization-evoked secretion requires two vicinal transmembrane cysteines of syntaxin 1A., Cohen R., PLoS One. December 5, 2007; 2 (12): e1273.
	The role of distal S6 hydrophobic residues in the voltage-dependent gating of CaV2.3 channels., Raybaud A., J Biol Chem. September 21, 2007; 282 (38): 27944-52.
	Co-localization and interaction of organic anion transporter 1 with caveolin-2 in rat kidney., Kwak JO., Exp Mol Med. June 30, 2005; 37 (3): 204-12.
	Overexpressed Ca(v)beta3 inhibits N-type (Cav2.2) calcium channel currents through a hyperpolarizing shift of ultra-slow and closed-state inactivation., Yasuda T., J Gen Physiol. April 1, 2004; 123 (4): 401-16.

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