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Comparison of the effects of antiarrhythmic drugs flecainide and verapamil on fKv1.4ΔN channel currents in Xenopus oocytes. , Chen H ., Acta Pharmacol Sin. February 1, 2013; 34 (2): 221-30.
Effects of diltiazem and propafenone on the inactivation and recovery kinetics of fKv1.4 channel currents expressed in Xenopus oocytes. , Zhang D., Acta Pharmacol Sin. April 1, 2011; 32 (4): 465-77.
Regulation of antiarrhythmic drug propafenone effects on the c-type Kv1.4 potassium channel by PHo and K+. , Wang Z., J Korean Med Sci. February 1, 2009; 24 (1): 84-91.
A single residue in the S6 transmembrane domain governs the differential flecainide sensitivity of voltage-gated potassium channels. , Herrera D., Mol Pharmacol. August 1, 2005; 68 (2): 305-16.
Inactivation and recovery in Kv1.4 K+ channels: lipophilic interactions at the intracellular mouth of the pore. , Bett GC., J Physiol. April 1, 2004; 556 (Pt 1): 109-20.
C-type inactivation involves a significant decrease in the intracellular aqueous pore volume of Kv1.4 K+ channels expressed in Xenopus oocytes. , Jiang X., J Physiol. June 15, 2003; 549 (Pt 3): 683-95.
Regulation of N- and C-type inactivation of Kv1.4 by pHo and K+: evidence for transmembrane communication. , Li X., Am J Physiol Heart Circ Physiol. January 1, 2003; 284 (1): H71-80.
Expression of voltage-dependent potassium channels in the developing visual system of Xenopus laevis. , Pollock NS., J Comp Neurol. October 28, 2002; 452 (4): 381-91.