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Summary Anatomy Item Literature (1949) Expression Attributions Wiki
XB-ANAT-3904

Papers associated with plasma membrane (and kcnj2)

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Bioelectric signalling via potassium channels: a mechanism for craniofacial dysmorphogenesis in KCNJ2-associated Andersen-Tawil Syndrome., Adams DS., J Physiol. June 15, 2016; 594 (12): 3245-70.                              


A splice variant of the two-pore domain potassium channel TREK-1 with only one pore domain reduces the surface expression of full-length TREK-1 channels., Rinné S., Pflugers Arch. August 1, 2014; 466 (8): 1559-70.


The bundle crossing region is responsible for the inwardly rectifying internal spermine block of the Kir2.1 channel., Huang CW., Pflugers Arch. February 1, 2014; 466 (2): 275-93.


Up-regulation of Kir2.1 (KCNJ2) by the serum & glucocorticoid inducible SGK3., Munoz C., Cell Physiol Biochem. January 1, 2014; 33 (2): 491-500.


Non dominant-negative KCNJ2 gene mutations leading to Andersen-Tawil syndrome with an isolated cardiac phenotype., Limberg MM., Basic Res Cardiol. May 1, 2013; 108 (3): 353.


Up-regulation of the inwardly rectifying K⁺ channel Kir2.1 (KCNJ2) by protein kinase B (PKB/Akt) and PIKfyve., Munoz C., J Membr Biol. March 1, 2013; 246 (3): 189-97.


Inhibition of Kir2.1 (KCNJ2) by the AMP-activated protein kinase., Alesutan I., Biochem Biophys Res Commun. May 20, 2011; 408 (4): 505-10.


Comparative analysis of cholesterol sensitivity of Kir channels: role of the CD loop., Rosenhouse-Dantsker A., Channels (Austin). January 1, 2010; 4 (1): 63-6.


KCNE1 and KCNE3 beta-subunits regulate membrane surface expression of Kv12.2 K(+) channels in vitro and form a tripartite complex in vivo., Clancy SM., PLoS One. July 22, 2009; 4 (7): e6330.          


Structural changes in the cytoplasmic pore of the Kir1.1 channel during pHi-gating probed by FRET., Lee JR., J Biomed Sci. March 6, 2009; 16 29.      


Functional and clinical characterization of a mutation in KCNJ2 associated with Andersen-Tawil syndrome., Lu CW., J Med Genet. August 1, 2006; 43 (8): 653-9.


betaL-betaM loop in the C-terminal domain of G protein-activated inwardly rectifying K(+) channels is important for G(betagamma) subunit activation., Finley M., J Physiol. March 16, 2004; 555 (Pt 3): 643-57.


Effect of isosorbiddinitrate on exogenously expressed slowly activating K+ channels and endogenous K+ channels in Xenopus oocytes., Busch AE., J Physiol. March 15, 1996; 491 ( Pt 3) 735-41.


Strong voltage-dependent inward rectification of inward rectifier K+ channels is caused by intracellular spermine., Fakler B., Cell. January 13, 1995; 80 (1): 149-54.


Cloning and expression of a novel human brain inward rectifier potassium channel., Makhina EN., J Biol Chem. August 12, 1994; 269 (32): 20468-74.

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