Papers associated with plasma membrane (and kcnb1)

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	Rearrangements in the relative orientation of cytoplasmic domains induced by a membrane-anchored protein mediate modulations in Kv channel gating., Lvov A., J Biol Chem. October 9, 2009; 284 (41): 28276-91.
	Dynamic coupling of voltage sensor and gate involved in closed-state inactivation of kv4.2 channels., Barghaan J., J Gen Physiol. February 1, 2009; 133 (2): 205-24.
	VAMP2 interacts directly with the N terminus of Kv2.1 to enhance channel inactivation., Lvov A., Pflugers Arch. September 1, 2008; 456 (6): 1121-36.
	Formation of the full SNARE complex eliminates interactions of its individual protein components with the Kv2.1 channel., Tsuk S., Biochemistry. August 12, 2008; 47 (32): 8342-9.
	Target soluble N-ethylmaleimide-sensitive factor attachment protein receptors (t-SNAREs) differently regulate activation and inactivation gating of Kv2.2 and Kv2.1: Implications on pancreatic islet cell Kv channels., Wolf-Goldberg T., Mol Pharmacol. September 1, 2006; 70 (3): 818-28.
	Kv2.1 channel activation and inactivation is influenced by physical interactions of both syntaxin 1A and the syntaxin 1A/soluble N-ethylmaleimide-sensitive factor-25 (t-SNARE) complex with the C terminus of the channel., Tsuk S., Mol Pharmacol. February 1, 2005; 67 (2): 480-8.
	Cyclic nucleotide-gated channels. Pore topology studied through the accessibility of reporter cysteines., Becchetti A., J Gen Physiol. September 1, 1999; 114 (3): 377-92.
	Modes of regulation of shab K+ channel activity by the Kv8.1 subunit., Salinas M., J Biol Chem. March 28, 1997; 272 (13): 8774-80.
	A new K+ channel beta subunit to specifically enhance Kv2.2 (CDRK) expression., Fink M., J Biol Chem. October 18, 1996; 271 (42): 26341-8.

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