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Summary Anatomy Item Literature (4897) Expression Attributions Wiki
XB-ANAT-3713

Papers associated with left (and orc2)

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Rif1 restrains the rate of replication origin firing in Xenopus laevis., Haccard O., Commun Biol. July 29, 2023; 6 (1): 788.                                


Differential nuclear import sets the timing of protein access to the embryonic genome., Nguyen T., Nat Commun. October 6, 2022; 13 (1): 5887.                                  


Polo-like kinase 1 (Plk1) regulates DNA replication origin firing and interacts with Rif1 in Xenopus., Ciardo D., Nucleic Acids Res. September 27, 2021; 49 (17): 9851-9869.                                        


Inhibition of DNA replication initiation by silver nanoclusters., Tao Y., Nucleic Acids Res. May 21, 2021; 49 (9): 5074-5083.                        


The High-Affinity Interaction between ORC and DNA that Is Required for Replication Licensing Is Inhibited by 2-Arylquinolin-4-Amines., Gardner NJ., Cell Chem Biol. August 17, 2017; 24 (8): 981-992.e4.                        


Depletion of Uhrf1 inhibits chromosomal DNA replication in Xenopus egg extracts., Taylor EM., Nucleic Acids Res. September 1, 2013; 41 (16): 7725-37.              


Treslin collaborates with TopBP1 in triggering the initiation of DNA replication., Kumagai A., Cell. February 5, 2010; 140 (3): 349-59.                        


The role of Cdc6 in ensuring complete genome licensing and S phase checkpoint activation., Oehlmann M., J Cell Biol. April 26, 2004; 165 (2): 181-90.                  


The Xenopus Xmus101 protein is required for the recruitment of Cdc45 to origins of DNA replication., Van Hatten RA., J Cell Biol. November 25, 2002; 159 (4): 541-7.          


Cyclin E uses Cdc6 as a chromatin-associated receptor required for DNA replication., Furstenthal L., J Cell Biol. March 19, 2001; 152 (6): 1267-78.                    


A role for Cdk2 kinase in negatively regulating DNA replication during S phase of the cell cycle., Hua XH., J Cell Biol. April 7, 1997; 137 (1): 183-92.            

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