Papers associated with ganglion (and kcnk18)

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	TRESK and TREK-2 two-pore-domain potassium channel subunits form functional heterodimers in primary somatosensory neurons., Lengyel M., J Biol Chem. August 28, 2020; 295 (35): 12408-12425.
	Chemically Modified Derivatives of the Activator Compound Cloxyquin Exert Inhibitory Effect on TRESK (K2P18.1) Background Potassium Channel., Lengyel M., Mol Pharmacol. June 1, 2019; 95 (6): 652-660.
	Selective and state-dependent activation of TRESK (K2P 18.1) background potassium channel by cloxyquin., Lengyel M., Br J Pharmacol. July 1, 2017; 174 (13): 2102-2113.
	Properties, regulation, pharmacology, and functions of the K₂p channel, TRESK., Enyedi P., Pflugers Arch. May 1, 2015; 467 (5): 945-58.
	Differential sensitivity of TREK-1, TREK-2 and TRAAK background potassium channels to the polycationic dye ruthenium red., Braun G., Br J Pharmacol. April 1, 2015; 172 (7): 1728-38.
	Activation of TRESK channels by the inflammatory mediator lysophosphatidic acid balances nociceptive signalling., Kollert S., Sci Rep. January 12, 2015; 5 12548.
	Nonmigraine-associated TRESK K+ channel variant C110R does not increase the excitability of trigeminal ganglion neurons., Guo Z., J Neurophysiol. August 1, 2014; 112 (3): 568-79.
	Functional characterization of zebrafish K2P18.1 (TRESK) two-pore-domain K+ channels., Rahm AK., Naunyn Schmiedebergs Arch Pharmacol. March 1, 2014; 387 (3): 291-300.
	Functional analysis of a migraine-associated TRESK K+ channel mutation., Liu P., J Neurosci. July 31, 2013; 33 (31): 12810-24.
	TRESK two-pore-domain K+ channels constitute a significant component of background potassium currents in murine dorsal root ganglion neurones., Dobler T., J Physiol. December 15, 2007; 585 (Pt 3): 867-79.

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