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Gli protein activity is controlled by multisite phosphorylation in vertebrate Hedgehog signaling. , Niewiadomski P., Cell Rep. January 16, 2014; 6 (1): 168-81.
Stabilization of speckle-type POZ protein ( Spop) by Daz interacting protein 1 ( Dzip1) is essential for Gli turnover and the proper output of Hedgehog signaling. , Schwend T ., J Biol Chem. November 8, 2013; 288 (45): 32809-32820.
Characterization of the hypothalamus of Xenopus laevis during development. I. The alar regions. , Domínguez L., J Comp Neurol. March 1, 2013; 521 (4): 725-59.
Live imaging of targeted cell ablation in Xenopus: a new model to study demyelination and repair. , Kaya F., J Neurosci. September 12, 2012; 32 (37): 12885-95.
A revised model of Xenopus dorsal midline development: differential and separable requirements for Notch and Shh signaling. , Peyrot SM., Dev Biol. April 15, 2011; 352 (2): 254-66.
The amino-terminal region of Gli3 antagonizes the Shh response and acts in dorsoventral fate specification in the developing spinal cord. , Meyer NP., Dev Biol. May 15, 2003; 257 (2): 343-55.
The morphogen sonic hedgehog is an axonal chemoattractant that collaborates with netrin-1 in midline axon guidance. , Charron F., Cell. April 4, 2003; 113 (1): 11-23.
Pax6 controls progenitor cell identity and neuronal fate in response to graded Shh signaling. , Ericson J., Cell. July 11, 1997; 90 (1): 169-80.