Papers associated with cardiac mesoderm (and kcnj2)

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	Class III antiarrhythmic drug dronedarone inhibits cardiac inwardly rectifying Kir2.1 channels through binding at residue E224., Xynogalos P., Naunyn Schmiedebergs Arch Pharmacol. December 1, 2014; 387 (12): 1153-61.
	Genetically induced dysfunctions of Kir2.1 channels: implications for short QT3 syndrome and autism-epilepsy phenotype., Ambrosini E., Hum Mol Genet. September 15, 2014; 23 (18): 4875-86.
	A Kir3.4 mutation causes Andersen-Tawil syndrome by an inhibitory effect on Kir2.1., Kokunai Y., Neurology. March 25, 2014; 82 (12): 1058-64.
	Inhibition of G protein-activated inwardly rectifying K+ channels by different classes of antidepressants., Kobayashi T., PLoS One. January 1, 2011; 6 (12): e28208.
	Inhibition by cocaine of G protein-activated inwardly rectifying K+ channels expressed in Xenopus oocytes., Kobayashi T., Toxicol In Vitro. June 1, 2007; 21 (4): 656-64.
	Functional and clinical characterization of a mutation in KCNJ2 associated with Andersen-Tawil syndrome., Lu CW., J Med Genet. August 1, 2006; 43 (8): 653-9.
	Polymorphic ventricular tachycardia and KCNJ2 mutations., Chun TU., Heart Rhythm. July 1, 2004; 1 (2): 235-41.
	Inhibition of G protein-activated inwardly rectifying K+ channels by fluoxetine (Prozac)., Kobayashi T., Br J Pharmacol. March 1, 2003; 138 (6): 1119-28.
	Mutations in Kir2.1 cause the developmental and episodic electrical phenotypes of Andersen's syndrome., Plaster NM., Cell. May 18, 2001; 105 (4): 511-9.
	Molecular characterization of an inwardly rectifying K+ channel from HeLa cells., Klein H., J Membr Biol. January 1, 1999; 167 (1): 43-52.
	Molecular and functional heterogeneity of inward rectifier potassium channels in brain and heart., Kurachi Y., J Card Fail. December 1, 1996; 2 (4 Suppl): S59-62.
	Susceptibility of cloned K+ channels to reactive oxygen species., Duprat F., Proc Natl Acad Sci U S A. December 5, 1995; 92 (25): 11796-800.
	Cloning and functional characterization of a novel ATP-sensitive potassium channel ubiquitously expressed in rat tissues, including pancreatic islets, pituitary, skeletal muscle, and heart., Inagaki N., J Biol Chem. March 17, 1995; 270 (11): 5691-4.
	Cloning and functional expression of an inwardly rectifying K+ channel from human atrium., Wible BA., Circ Res. March 1, 1995; 76 (3): 343-50.
	Cloning, localization, and functional expression of a human brain inward rectifier potassium channel (hIRK1)., Tang W., Recept Channels. January 1, 1995; 3 (3): 175-83.
	Molecular cloning and expression of a human heart inward rectifier potassium channel., Raab-Graham KF., Neuroreport. December 20, 1994; 5 (18): 2501-5.
	Gating mechanism of the cloned inward rectifier potassium channel from mouse heart., Ishihara K., J Membr Biol. October 1, 1994; 142 (1): 55-64.
	Molecular cloning and functional expression of cDNA encoding a second class of inward rectifier potassium channels in the mouse brain., Takahashi N., J Biol Chem. September 16, 1994; 269 (37): 23274-9.
	Cloning a novel human brain inward rectifier potassium channel and its functional expression in Xenopus oocytes., Tang W., FEBS Lett. July 18, 1994; 348 (3): 239-43.
	Cloning and functional expression of a cardiac inward rectifier K+ channel., Ishii K., FEBS Lett. January 24, 1994; 338 (1): 107-11.
	Primary structure and functional expression of a rat G-protein-coupled muscarinic potassium channel., Kubo Y., Nature. August 26, 1993; 364 (6440): 802-6.

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