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Bioelectric signalling via potassium channels: a mechanism for craniofacial dysmorphogenesis in KCNJ2-associated Andersen-Tawil Syndrome. , Adams DS ., J Physiol. June 15, 2016; 594 (12): 3245-70.
Distinct action of the α-glucosidase inhibitor miglitol on SGLT3, enteroendocrine cells, and GLP1 secretion. , Lee EY ., J Endocrinol. March 1, 2015; 224 (3): 205-14.
A universally conserved residue in the SUR1 subunit of the KATP channel is essential for translating nucleotide binding at SUR1 into channel opening. , de Wet H., J Physiol. October 15, 2012; 590 (20): 5025-36.
Mutations of the same conserved glutamate residue in NBD2 of the sulfonylurea receptor 1 subunit of the KATP channel can result in either hyperinsulinism or neonatal diabetes. , Männikkö R., Diabetes. June 1, 2011; 60 (6): 1813-22.
Activation of the K(ATP) channel by Mg-nucleotide interaction with SUR1. , Proks P., J Gen Physiol. October 1, 2010; 136 (4): 389-405.
A Kir6.2 mutation causing severe functional effects in vitro produces neonatal diabetes without the expected neurological complications. , Tammaro P., Diabetologia. May 1, 2008; 51 (5): 802-10.