Papers associated with oocyte (and gjb2)

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	Divergence between Hemichannel and Gap Junction Permeabilities of Connexin 30 and 26., Xu J., Life (Basel). January 31, 2023; 13 (2):
	PACmn for improved optogenetic control of intracellular cAMP., Yang S., BMC Biol. October 18, 2021; 19 (1): 227.
	A novel voltage-clamp/dye uptake assay reveals saturable transport of molecules through CALHM1 and connexin channels., Gaete PS., J Gen Physiol. November 2, 2020; 152 (11):
	Calmodulin-Cork Model of Gap Junction Channel Gating-One Molecule, Two Mechanisms., Peracchia C., Int J Mol Sci. July 13, 2020; 21 (14):
	S-nitrosylation of connexin43 hemichannels elicits cardiac stress-induced arrhythmias in Duchenne muscular dystrophy mice., Lillo MA., JCI Insight. December 19, 2019; 4 (24):
	Structural determinants underlying permeant discrimination of the Cx43 hemichannel., Nielsen BS., J Biol Chem. November 8, 2019; 294 (45): 16789-16803.
	The connexin26 human mutation N14K disrupts cytosolic intersubunit interactions and promotes channel opening., Valdez Capuccino JM., J Gen Physiol. March 4, 2019; 151 (3): 328-341.
	A novel autosomal recessive GJB2-associated disorder: Ichthyosis follicularis, bilateral severe sensorineural hearing loss, and punctate palmoplantar keratoderma., Youssefian L., Hum Mutat. February 1, 2019; 40 (2): 217-229.
	Syndromic deafness mutations at Asn 14 differentially alter the open stability of Cx26 hemichannels., Sanchez HA., J Gen Physiol. July 1, 2016; 148 (1): 25-42.
	Glutathione release through connexin hemichannels: Implications for chemical modification of pores permeable to large molecules., Tong X., J Gen Physiol. September 1, 2015; 146 (3): 245-54.
	The comprehensive analysis of DEG/ENaC subunits in Hydra reveals a large variety of peptide-gated channels, potentially involved in neuromuscular transmission., Assmann M., BMC Biol. October 14, 2014; 12 84.
	The D50N mutation and syndromic deafness: altered Cx26 hemichannel properties caused by effects on the pore and intersubunit interactions., Sanchez HA., J Gen Physiol. July 1, 2013; 142 (1): 3-22.
	Voltage-dependent gating of the Cx32*43E1 hemichannel: conformational changes at the channel entrances., Kwon T., J Gen Physiol. February 1, 2013; 141 (2): 243-59.
	Analysis of trafficking, stability and function of human connexin 26 gap junction channels with deafness-causing mutations in the fourth transmembrane helix., Ambrosi C., PLoS One. January 1, 2013; 8 (8): e70916.
	Mechanism of inhibition of connexin channels by the quinine derivative N-benzylquininium., Rubinos C., J Gen Physiol. January 1, 2012; 139 (1): 69-82.
	Inhibition of connexin 26 by the AMP-activated protein kinase., Alesutan I., J Membr Biol. April 1, 2011; 240 (3): 151-8.
	Pannexin1 and Pannexin2 channels show quaternary similarities to connexons and different oligomerization numbers from each other., Ambrosi C., J Biol Chem. August 6, 2010; 285 (32): 24420-31.
	Differentially altered Ca2+ regulation and Ca2+ permeability in Cx26 hemichannels formed by the A40V and G45E mutations that cause keratitis ichthyosis deafness syndrome., Sánchez HA., J Gen Physiol. July 1, 2010; 136 (1): 47-62.
	Aberrant hemichannel properties of Cx26 mutations causing skin disease and deafness., Gerido DA., Am J Physiol Cell Physiol. July 1, 2007; 293 (1): C337-45.
	Properties of connexin26 hemichannels expressed in Xenopus oocytes., Ripps H., Cell Mol Neurobiol. October 1, 2004; 24 (5): 647-65.
	A novel connexin 26 gene mutation associated with features of the keratitis-ichthyosis-deafness syndrome and the follicular occlusion triad., Montgomery JR., J Am Acad Dermatol. September 1, 2004; 51 (3): 377-82.
	The permeability of gap junction channels to probes of different size is dependent on connexin composition and permeant-pore affinities., Weber PA., Biophys J. August 1, 2004; 87 (2): 958-73.
	Altered gating properties of functional Cx26 mutants associated with recessive non-syndromic hearing loss., Meşe G., Hum Genet. August 1, 2004; 115 (3): 191-9.
	Functional analysis of a dominant mutation of human connexin26 associated with nonsyndromic deafness., Bruzzone R., Cell Commun Adhes. January 1, 2001; 8 (4-6): 425-31.
	Size selectivity between gap junction channels composed of different connexins., Gong XQ., Cell Commun Adhes. January 1, 2001; 8 (4-6): 187-92.
	Stoichiometry of transjunctional voltage-gating polarity reversal by a negative charge substitution in the amino terminus of a connexin32 chimera., Oh S., J Gen Physiol. July 1, 2000; 116 (1): 13-31.
	Functional analysis of human Cx26 mutations associated with deafness., White TW., Brain Res Brain Res Rev. April 1, 2000; 32 (1): 181-3.
	Hetero-domain interactions as a mechanism for the regulation of connexin channels., Stergiopoulos K., Circ Res. May 28, 1999; 84 (10): 1144-55.
	Connexin32 mutations associated with X-linked Charcot-Marie-Tooth disease show two distinct behaviors: loss of function and altered gating properties., Ressot C., J Neurosci. June 1, 1998; 18 (11): 4063-75.
	A quantitative analysis of connexin-specific permeability differences of gap junctions expressed in HeLa transfectants and Xenopus oocytes., Cao F., J Cell Sci. January 1, 1998; 111 ( Pt 1) 31-43.
	Gap junctions formed by connexins 26 and 32 alone and in combination are differently affected by applied voltage., Barrio LC., Proc Natl Acad Sci U S A. October 1, 1991; 88 (19): 8410-4.

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