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Inherited macular degeneration-associated mutations in CNGB3 increase the ligand sensitivity and spontaneous open probability of cone cyclic nucleotide-gated channels. , Meighan PC., Front Physiol. June 9, 2015; 6 177.
Two structural components in CNGA3 support regulation of cone CNG channels by phosphoinositides. , Dai G., J Gen Physiol. April 1, 2013; 141 (4): 413-30.
Matrix metalloproteinase-9 and -2 enhance the ligand sensitivity of photoreceptor cyclic nucleotide-gated channels. , Meighan PC., Channels (Austin). January 1, 2012; 6 (3): 181-96.
Regulation of human cone cyclic nucleotide-gated channels by endogenous phospholipids and exogenously applied phosphatidylinositol 3,4,5-trisphosphate. , Bright SR., Mol Pharmacol. January 1, 2007; 71 (1): 176-83.
Disease-associated mutations in CNGB3 produce gain of function alterations in cone cyclic nucleotide-gated channels. , Bright SR., Mol Vis. December 19, 2005; 11 1141-50.
Achromatopsia-associated mutation in the human cone photoreceptor cyclic nucleotide-gated channel CNGB3 subunit alters the ligand sensitivity and pore properties of heteromeric channels. , Peng C., J Biol Chem. September 5, 2003; 278 (36): 34533-40.
Functionally important calmodulin-binding sites in both NH2- and COOH-terminal regions of the cone photoreceptor cyclic nucleotide-gated channel CNGB3 subunit. , Peng C., J Biol Chem. July 4, 2003; 278 (27): 24617-23.