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Identification of Crucial Residues in α-Conotoxin EI Inhibiting Muscle Nicotinic Acetylcholine Receptor. , Ning J., Toxins (Basel). October 16, 2019; 11 (10):
6-bromohypaphorine from marine nudibranch mollusk Hermissenda crassicornis is an agonist of human α7 nicotinic acetylcholine receptor. , Kasheverov IE., Mar Drugs. March 12, 2015; 13 (3): 1255-66.
Structural and gating changes of the sodium channel induced by mutation of a residue in the upper third of IVS6, creating an external access path for local anesthetics. , Sunami A., Mol Pharmacol. April 1, 2001; 59 (4): 684-91.
Regulation of Shaker-type potassium channels by hypoxia. Oxygen-sensitive K+ channels in PC12 cells. , Conforti L., Adv Exp Med Biol. January 1, 2000; 475 265-74.
Extrapore residues of the S5-S6 loop of domain 2 of the voltage-gated skeletal muscle sodium channel (rSkM1) contribute to the mu-conotoxin GIIIA binding site. , Chahine M., Biophys J. July 1, 1998; 75 (1): 236-46.
Pore residues critical for mu- CTX binding to rat skeletal muscle Na+ channels revealed by cysteine mutagenesis. , Li RA., Biophys J. October 1, 1997; 73 (4): 1874-84.
Characterizing the mu-conotoxin binding site on voltage-sensitive sodium channels with toxin analogs and channel mutations. , Chahine M., Recept Channels. January 1, 1995; 3 (3): 161-74.
Heterotetramer formation and charybdotoxin sensitivity of two K+ channels cloned from smooth muscle. , Russell SN., Am J Physiol. December 1, 1994; 267 (6 Pt 1): C1729-33.
Chimeric study of sodium channels from rat skeletal and cardiac muscle. , Chen LQ., FEBS Lett. September 14, 1992; 309 (3): 253-7.