Papers associated with anatomical group (and kcnj1)

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	Aquaporin-2: new mutations responsible for autosomal-recessive nephrogenic diabetes insipidus-update and epidemiology., Bichet DG., Clin Kidney J. June 1, 2012; 5 (3): 195-202.
	Ion selectivity and current saturation in inward-rectifier K+ channels., Yang L., J Gen Physiol. February 1, 2012; 139 (2): 145-57.
	Functional analysis of Rfx6 and mutant variants associated with neonatal diabetes., Pearl EJ., Dev Biol. March 1, 2011; 351 (1): 135-45.
	Inhibition of g protein-activated inwardly rectifying k channels by phencyclidine., Kobayashi T., Curr Neuropharmacol. March 1, 2011; 9 (1): 244-6.
	Pregnenolone sulfate potentiates the inwardly rectifying K channel Kir2.3., Kobayashi T., PLoS One. July 21, 2009; 4 (7): e6311.
	Dietary K regulates ROMK channels in connecting tubule and cortical collecting duct of rat kidney., Frindt G., Am J Physiol Renal Physiol. February 1, 2009; 296 (2): F347-54.
	Long polyamines act as cofactors in PIP2 activation of inward rectifier potassium (Kir2.1) channels., Xie LH., J Gen Physiol. December 1, 2005; 126 (6): 541-9.
	Basolateral K+ conductance in principal cells of rat CCD., Gray DA., Am J Physiol Renal Physiol. March 1, 2005; 288 (3): F493-504.
	Expression and coexpression of CO2-sensitive Kir channels in brainstem neurons of rats., Wu J., J Membr Biol. February 1, 2004; 197 (3): 179-91.
	Barttin is a Cl- channel beta-subunit crucial for renal Cl- reabsorption and inner ear K+ secretion., Estévez R., Nature. November 29, 2001; 414 (6863): 558-61.
	Mechanism underlying bupivacaine inhibition of G protein-gated inwardly rectifying K+ channels., Zhou W., Proc Natl Acad Sci U S A. May 22, 2001; 98 (11): 6482-7.
	A single residue contributes to the difference between Kir4.1 and Kir1.1 channels in pH sensitivity, rectification and single channel conductance., Xu H., J Physiol. October 15, 2000; 528 Pt 2 267-77.
	Probing the water permeability of ROMK1 and amphotericin B channels using Xenopus oocytes., Sabirov RZ., Biochim Biophys Acta. January 5, 1998; 1368 (1): 19-26.
	A novel ATP-dependent inward rectifier potassium channel expressed predominantly in glial cells., Takumi T., J Biol Chem. July 7, 1995; 270 (27): 16339-46.
	Cloning provides evidence for a family of inward rectifier and G-protein coupled K+ channels in the brain., Lesage F., FEBS Lett. October 10, 1994; 353 (1): 37-42.
	Molecular cloning, functional expression and localization of a novel inward rectifier potassium channel in the rat brain., Koyama H., FEBS Lett. March 21, 1994; 341 (2-3): 303-7.

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