Papers associated with brain (and kcnj5)

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	A revised mechanism of action of hyperaldosteronism-linked mutations in cytosolic domains of GIRK4 (KCNJ5)., Shalomov B., J Physiol. March 1, 2022; 600 (6): 1419-1437.
	The small molecule GAT1508 activates brain-specific GIRK1/2 channel heteromers and facilitates conditioned fear extinction in rodents., Xu Y., J Biol Chem. March 13, 2020; 295 (11): 3614-3634.
	The GIRK1 brain variant GIRK1d and its functional impact on heteromultimeric GIRK channels., Steinecker B., J Recept Signal Transduct Res. January 1, 2007; 27 (5-6): 369-82.
	RFamide-related peptides signal through the neuropeptide FF receptor and regulate pain-related responses in the rat., Pertovaara A., Neuroscience. January 1, 2005; 134 (3): 1023-32.
	Single channel analysis of the regulation of GIRK1/GIRK4 channels by protein phosphorylation., Müllner D., Biophys J. February 1, 2003; 84 (2 Pt 1): 1399-409.
	Early embryonic expression of ion channels and pumps in chick and Xenopus development., Rutenberg J., Dev Dyn. December 1, 2002; 225 (4): 469-84.
	N-terminal tyrosine residues within the potassium channel Kir3 modulate GTPase activity of Galphai., Ippolito DL., J Biol Chem. September 6, 2002; 277 (36): 32692-6.
	Functional expression of a novel ginsenoside Rf binding protein from rat brain mRNA in Xenopus laevis oocytes., Choi S., Mol Pharmacol. April 1, 2002; 61 (4): 928-35.
	Identification of G protein-coupled, inward rectifier potassium channel gene products from the rat anterior pituitary gland., Gregerson KA., Endocrinology. July 1, 2001; 142 (7): 2820-32.
	G-protein mediated gating of inward-rectifier K+ channels., Mark MD., Eur J Biochem. October 1, 2000; 267 (19): 5830-6.
	TrkB activation by brain-derived neurotrophic factor inhibits the G protein-gated inward rectifier Kir3 by tyrosine phosphorylation of the channel., Rogalski SL., J Biol Chem. August 18, 2000; 275 (33): 25082-8.
	Inhibition by various antipsychotic drugs of the G-protein-activated inwardly rectifying K(+) (GIRK) channels expressed in xenopus oocytes., Kobayashi T., Br J Pharmacol. April 1, 2000; 129 (8): 1716-22.
	Functional expression and characterization of G-protein-gated inwardly rectifying K+ channels containing GIRK3., Jelacic TM., J Membr Biol. May 15, 1999; 169 (2): 123-9.
	Cloned human and rat galanin GALR3 receptors. Pharmacology and activation of G-protein inwardly rectifying K+ channels., Smith KE., J Biol Chem. September 4, 1998; 273 (36): 23321-6.
	A regenerative link in the ionic fluxes through the weaver potassium channel underlies the pathophysiology of the mutation., Silverman SK., Proc Natl Acad Sci U S A. December 24, 1996; 93 (26): 15429-34.
	Control of channel activity through a unique amino acid residue of a G protein-gated inwardly rectifying K+ channel subunit., Chan KW., Proc Natl Acad Sci U S A. November 26, 1996; 93 (24): 14193-8.
	Specificity of coupling of muscarinic receptor isoforms to a novel chick inward-rectifying acetylcholine-sensitive K+ channel., Gadbut AP., J Biol Chem. March 15, 1996; 271 (11): 6398-402.
	A G-protein-activated inwardly rectifying K+ channel (GIRK4) from human hippocampus associates with other GIRK channels., Spauschus A., J Neurosci. February 1, 1996; 16 (3): 930-8.
	The K+ channel inward rectifier subunits form a channel similar to neuronal G protein-gated K+ channel., Velimirovic BM., FEBS Lett. January 22, 1996; 379 (1): 31-7.

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