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Summary Anatomy Item Literature (6354) Expression Attributions Wiki
XB-ANAT-254

Papers associated with oocyte (and kcna4)

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S-glutathionylation of an auxiliary subunit confers redox sensitivity to Kv4 channel inactivation., Jerng HH., PLoS One. January 1, 2014; 9 (3): e93315.                      

C-type inactivation involves a significant decrease in the intracellular aqueous pore volume of Kv1.4 K+ channels expressed in Xenopus oocytes., Jiang X., J Physiol. June 15, 2003; 549 (Pt 3): 683-95.

Inactivation gating of Kv4 potassium channels: molecular interactions involving the inner vestibule of the pore., Jerng HH., J Gen Physiol. May 1, 1999; 113 (5): 641-60.                        

Toxin and subunit specificity of blocking affinity of three peptide toxins for heteromultimeric, voltage-gated potassium channels expressed in Xenopus oocytes., Hopkins WF., J Pharmacol Exp Ther. June 1, 1998; 285 (3): 1051-60.

Antiarrhythmic and bradycardic drugs inhibit currents of cloned K+ channels, KV1.2 and KV1.4., Yamagishi T., Eur J Pharmacol. August 4, 1995; 281 (2): 151-9.

Time- and voltage-dependent modulation of a Kv1.4 channel by a beta-subunit (Kv beta 3) cloned from ferret ventricle., Castellino RC., Am J Physiol. July 1, 1995; 269 (1 Pt 2): H385-91.

Bi-stable block by 4-aminopyridine of a transient K+ channel (Kv1.4) cloned from ferret ventricle and expressed in Xenopus oocytes., Rasmusson RL., J Physiol. May 15, 1995; 485 ( Pt 1) 59-71.

Absence of effects of class III antiarrhythmic agents on cloned cardiac K channels., Yamagishi T., Jpn J Pharmacol. April 1, 1993; 61 (4): 371-3.

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