Papers associated with egg (and rad50)

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	Resection of DNA double-strand breaks activates Mre11-Rad50-Nbs1- and Rad9-Hus1-Rad1-dependent mechanisms that redundantly promote ATR checkpoint activation and end processing in Xenopus egg extracts., Tatsukawa K., Nucleic Acids Res. April 12, 2024; 52 (6): 3146-3163.
	MRN-dependent and independent pathways for recruitment of TOPBP1 to DNA double-strand breaks., Montales K., PLoS One. August 2, 2022; 17 (8): e0271905.
	Mdc1 modulates the interaction between TopBP1 and the MRN complex during DNA damage checkpoint responses., Choi SH., Biochem Biophys Res Commun. October 7, 2016; 479 (1): 5-11.
	MRN, CtIP, and BRCA1 mediate repair of topoisomerase II-DNA adducts., Aparicio T., J Cell Biol. February 15, 2016; 212 (4): 399-408.
	The Mre11-Rad50-Nbs1 (MRN) complex has a specific role in the activation of Chk1 in response to stalled replication forks., Lee J., Mol Biol Cell. May 1, 2013; 24 (9): 1343-53.
	A role for the MRN complex in ATR activation via TOPBP1 recruitment., Duursma AM., Mol Cell. April 11, 2013; 50 (1): 116-22.
	The MRN-CtIP pathway is required for metaphase chromosome alignment., Rozier L., Mol Cell. March 28, 2013; 49 (6): 1097-107.
	Analysis of MRE11's function in the 5'-->3' processing of DNA double-strand breaks., Liao S., Nucleic Acids Res. May 1, 2012; 40 (10): 4496-506.
	Role for Rif1 in the checkpoint response to damaged DNA in Xenopus egg extracts., Kumar S., Cell Cycle. March 15, 2012; 11 (6): 1183-94.
	CtIP interacts with TopBP1 and Nbs1 in the response to double-stranded DNA breaks (DSBs) in Xenopus egg extracts., Ramírez-Lugo JS., Cell Cycle. February 1, 2011; 10 (3): 469-80.
	The Mre11/Rad50/Nbs1 complex functions in resection-based DNA end joining in Xenopus laevis., Taylor EM., Nucleic Acids Res. January 1, 2010; 38 (2): 441-54.
	CtIP links DNA double-strand break sensing to resection., You Z., Mol Cell. December 25, 2009; 36 (6): 954-69.
	The Mre11-Rad50-Nbs1 complex mediates activation of TopBP1 by ATM., Yoo HY., Mol Biol Cell. May 1, 2009; 20 (9): 2351-60.
	Mre11-Rad50-Nbs1-dependent processing of DNA breaks generates oligonucleotides that stimulate ATM activity., Jazayeri A., EMBO J. July 23, 2008; 27 (14): 1953-62.
	ATM and ATR promote Mre11 dependent restart of collapsed replication forks and prevent accumulation of DNA breaks., Trenz K., EMBO J. April 19, 2006; 25 (8): 1764-74.
	Repair of double-strand breaks by nonhomologous end joining in the absence of Mre11., Di Virgilio M., J Cell Biol. December 5, 2005; 171 (5): 765-71.
	ATM activation and its recruitment to damaged DNA require binding to the C terminus of Nbs1., You Z., Mol Cell Biol. July 1, 2005; 25 (13): 5363-79.

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