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Resection of DNA double-strand breaks activates Mre11- Rad50- Nbs1- and Rad9- Hus1- Rad1-dependent mechanisms that redundantly promote ATR checkpoint activation and end processing in Xenopus egg extracts. , Tatsukawa K., Nucleic Acids Res. April 12, 2024; 52 (6): 3146-3163.
POLθ prevents MRE11- NBS1- CtIP-dependent fork breakage in the absence of BRCA2/RAD51 by filling lagging-strand gaps. , Mann A., Mol Cell. November 17, 2022; 82 (22): 4218-4231.e8.
MRN-dependent and independent pathways for recruitment of TOPBP1 to DNA double-strand breaks. , Montales K., PLoS One. August 2, 2022; 17 (8): e0271905.
The structure of ends determines the pathway choice and Mre11 nuclease dependency of DNA double-strand break repair. , Liao S., Nucleic Acids Res. July 8, 2016; 44 (12): 5689-701.
MRN, CtIP, and BRCA1 mediate repair of topoisomerase II-DNA adducts. , Aparicio T ., J Cell Biol. February 15, 2016; 212 (4): 399-408.
The MRN- CtIP pathway is required for metaphase chromosome alignment. , Rozier L., Mol Cell. March 28, 2013; 49 (6): 1097-107.
Analysis of MRE11's function in the 5'-->3' processing of DNA double-strand breaks. , Liao S., Nucleic Acids Res. May 1, 2012; 40 (10): 4496-506.
Time-dependent predominance of nonhomologous DNA end-joining pathways during embryonic development in mice. , Chiruvella KK., J Mol Biol. March 30, 2012; 417 (3): 197-211.
Cdk1 uncouples CtIP-dependent resection and Rad51 filament formation during M-phase double-strand break repair. , Peterson SE., J Cell Biol. September 5, 2011; 194 (5): 705-20.
CtIP interacts with TopBP1 and Nbs1 in the response to double-stranded DNA breaks (DSBs) in Xenopus egg extracts. , Ramírez-Lugo JS., Cell Cycle. February 1, 2011; 10 (3): 469-80.
Replication protein A promotes 5'-->3' end processing during homology-dependent DNA double-strand break repair. , Yan H., J Cell Biol. January 24, 2011; 192 (2): 251-61.
CtIP links DNA double-strand break sensing to resection. , You Z., Mol Cell. December 25, 2009; 36 (6): 954-69.