Papers associated with egg (and exo1)

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	Resection of DNA double-strand breaks activates Mre11-Rad50-Nbs1- and Rad9-Hus1-Rad1-dependent mechanisms that redundantly promote ATR checkpoint activation and end processing in Xenopus egg extracts., Tatsukawa K., Nucleic Acids Res. April 12, 2024; 52 (6): 3146-3163.
	Crosstalk between repair pathways elicits double-strand breaks in alkylated DNA and implications for the action of temozolomide., Fuchs RP., Elife. July 8, 2021; 10
	HPF1-dependent PARP activation promotes LIG3-XRCC1-mediated backup pathway of Okazaki fragment ligation., Kumamoto S., Nucleic Acids Res. May 21, 2021; 49 (9): 5003-5016.
	The structure of ends determines the pathway choice and Mre11 nuclease dependency of DNA double-strand break repair., Liao S., Nucleic Acids Res. July 8, 2016; 44 (12): 5689-701.
	DNA double-strand breaks with 5' adducts are efficiently channeled to the DNA2-mediated resection pathway., Tammaro M., Nucleic Acids Res. January 8, 2016; 44 (1): 221-31.
	Poly(ADP-ribose)-binding promotes Exo1 damage recruitment and suppresses its nuclease activities., Cheruiyot A., DNA Repair (Amst). November 1, 2015; 35 106-15.
	The N-terminus of RPA large subunit and its spatial position are important for the 5'->3' resection of DNA double-strand breaks., Tammaro M., Nucleic Acids Res. October 15, 2015; 43 (18): 8790-800.
	14-3-3 proteins restrain the Exo1 nuclease to prevent overresection., Chen X., J Biol Chem. May 8, 2015; 290 (19): 12300-12.
	Analysis of MRE11's function in the 5'-->3' processing of DNA double-strand breaks., Liao S., Nucleic Acids Res. May 1, 2012; 40 (10): 4496-506.
	Cdk1 uncouples CtIP-dependent resection and Rad51 filament formation during M-phase double-strand break repair., Peterson SE., J Cell Biol. September 5, 2011; 194 (5): 705-20.

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