Papers associated with embryo (and invs)

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	Modeling congenital kidney diseases in Xenopus laevis., Blackburn ATM., Dis Model Mech. April 9, 2019; 12 (4):
	The ciliopathy-associated CPLANE proteins direct basal body recruitment of intraflagellar transport machinery., Toriyama M., Nat Genet. June 1, 2016; 48 (6): 648-56.
	Using Xenopus to study genetic kidney diseases., Lienkamp SS., Semin Cell Dev Biol. March 1, 2016; 51 117-24.
	The Wnt/JNK signaling target gene alcam is required for embryonic kidney development., Cizelsky W., Development. May 1, 2014; 141 (10): 2064-74.
	ANKS6 is a central component of a nephronophthisis module linking NEK8 to INVS and NPHP3., Hoff S., Nat Genet. August 1, 2013; 45 (8): 951-6.
	Inversin relays Frizzled-8 signals to promote proximal pronephros development., Lienkamp S., Proc Natl Acad Sci U S A. November 23, 2010; 107 (47): 20388-93.
	Loss of nephrocystin-3 function can cause embryonic lethality, Meckel-Gruber-like syndrome, situs inversus, and renal-hepatic-pancreatic dysplasia., Bergmann C., Am J Hum Genet. April 1, 2008; 82 (4): 959-70.
	Cilia-driven leftward flow determines laterality in Xenopus., Schweickert A., Curr Biol. January 9, 2007; 17 (1): 60-6.
	Localization and loss-of-function implicates ciliary proteins in early, cytoplasmic roles in left-right asymmetry., Qiu D., Dev Dyn. September 1, 2005; 234 (1): 176-89.
	Inversin, the gene product mutated in nephronophthisis type II, functions as a molecular switch between Wnt signaling pathways., Simons M., Nat Genet. May 1, 2005; 37 (5): 537-43.
	The left-right determinant inversin has highly conserved ankyrin repeat and IQ domains and interacts with calmodulin., Morgan D., Hum Genet. April 1, 2002; 110 (4): 377-84.

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