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Time-resolved quantitative proteomic analysis of the developing Xenopus otic vesicle reveals putative congenital hearing loss candidates. , Baxi AB., iScience. September 15, 2023; 26 (9): 107665.
The CHARGE syndrome ortholog CHD-7 regulates TGF-β pathways in Caenorhabditis elegans. , Jofré DM., Proc Natl Acad Sci U S A. April 12, 2022; 119 (15): e2109508119.
A convergent molecular network underlying autism and congenital heart disease. , Rosenthal SB., Cell Syst. November 17, 2021; 12 (11): 1094-1107.e6.
Modeling human congenital disorders with neural crest developmental defects using patient-derived induced pluripotent stem cells. , Okuno H., Regen Ther. August 24, 2021; 18 275-280.
Using Xenopus to analyze neurocristopathies like Kabuki syndrome. , Schwenty-Lara J., Genesis. February 1, 2021; 59 (1-2): e23404.
The neural border: Induction, specification and maturation of the territory that generates neural crest cells. , Pla P., Dev Biol. December 1, 2018; 444 Suppl 1 S36-S46.
Xenopus as a model organism for birth defects-Congenital heart disease and heterotaxy. , Duncan AR., Semin Cell Dev Biol. March 1, 2016; 51 73-9.
CHD7, the gene mutated in CHARGE syndrome, regulates genes involved in neural crest cell guidance. , Schulz Y., Hum Genet. August 1, 2014; 133 (8): 997-1009.
sizzled function and secreted factor network dynamics. , Shi J., Biol Open. March 15, 2012; 1 (3): 286-94.
Snail2 controls mesodermal BMP/Wnt induction of neural crest. , Shi J., Development. August 1, 2011; 138 (15): 3135-45.
CHD7 cooperates with PBAF to control multipotent neural crest formation. , Bajpai R ., Nature. February 18, 2010; 463 (7283): 958-62.
The SNF2 domain protein family in higher vertebrates displays dynamic expression patterns in Xenopus laevis embryos. , Linder B., Gene. February 4, 2004; 326 59-66.