Papers associated with embryo (and nav1)

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	Prdm12 Directs Nociceptive Sensory Neuron Development by Regulating the Expression of the NGF Receptor TrkA., Desiderio S., Cell Rep. March 26, 2019; 26 (13): 3522-3536.e5.
	Electrophysiological characterization of a large set of novel variants in the SCN5A-gene: identification of novel LQTS3 and BrS mutations., Ortiz-Bonnin B., Pflugers Arch. August 1, 2016; 468 (8): 1375-87.
	Binary architecture of the Nav1.2-β2 signaling complex., Das S., Elife. January 28, 2016; 5
	Gain-of-function mutation in TASK-4 channels and severe cardiac conduction disorder., Friedrich C., EMBO Mol Med. July 1, 2014; 6 (7): 937-51.
	The endocannabinoid anandamide inhibits voltage-gated sodium channels Nav1.2, Nav1.6, Nav1.7, and Nav1.8 in Xenopus oocytes., Okura D., Anesth Analg. March 1, 2014; 118 (3): 554-62.
	Tuning voltage-gated channel activity and cellular excitability with a sphingomyelinase., Combs DJ., J Gen Physiol. October 1, 2013; 142 (4): 367-80.
	Domain IV voltage-sensor movement is both sufficient and rate limiting for fast inactivation in sodium channels., Capes DL., J Gen Physiol. August 1, 2013; 142 (2): 101-12.
	Molecular mechanism of voltage sensing in voltage-gated proton channels., Gonzalez C., J Gen Physiol. March 1, 2013; 141 (3): 275-85.
	Multiple pore conformations driven by asynchronous movements of voltage sensors in a eukaryotic sodium channel., Goldschen-Ohm MP., Nat Commun. January 1, 2013; 4 1350.
	Transmembrane voltage potential controls embryonic eye patterning in Xenopus laevis., Pai VP., Development. January 1, 2012; 139 (2): 313-23.
	A proton leak current through the cardiac sodium channel is linked to mixed arrhythmia and the dilated cardiomyopathy phenotype., Gosselin-Badaroudine P., PLoS One. January 1, 2012; 7 (5): e38331.
	Functional properties and toxin pharmacology of a dorsal root ganglion sodium channel viewed through its voltage sensors., Bosmans F., J Gen Physiol. July 1, 2011; 138 (1): 59-72.
	Pharmacological modulation of brain Nav1.2 and cardiac Nav1.5 subtypes by the local anesthetic ropivacaine., Cheng HW., Neurosci Bull. August 1, 2010; 26 (4): 289-96.
	Optogenetic localization and genetic perturbation of saccade-generating neurons in zebrafish., Schoonheim PJ., J Neurosci. May 19, 2010; 30 (20): 7111-20.
	Blocking effect of methylflavonolamine on human Na(V)1.5 channels expressed in Xenopus laevis oocytes and on sodium currents in rabbit ventricular myocytes., Fan XR., Acta Pharmacol Sin. March 1, 2010; 31 (3): 297-306.
	Correlations between clinical and physiological consequences of the novel mutation R878C in a highly conserved pore residue in the cardiac Na+ channel., Zhang Y., Acta Physiol (Oxf). December 1, 2008; 194 (4): 311-23.
	Nav channel mechanosensitivity: activation and inactivation accelerate reversibly with stretch., Morris CE., Biophys J. August 1, 2007; 93 (3): 822-33.
	GLUT8 is dispensable for embryonic development but influences hippocampal neurogenesis and heart function., Membrez M., Mol Cell Biol. June 1, 2006; 26 (11): 4268-76.
	RE-1 silencer of transcription/neural restrictive silencer factor modulates ectodermal patterning during Xenopus development., Olguín P., J Neurosci. March 8, 2006; 26 (10): 2820-9.
	Distribution and functional characterization of human Nav1.3 splice variants., Thimmapaya R., Eur J Neurosci. July 1, 2005; 22 (1): 1-9.
	Importance of the conserved aromatic residues in the scorpion alpha-like toxin BmK M1: the hydrophobic surface region revisited., Sun YM., J Biol Chem. June 27, 2003; 278 (26): 24125-31.
	Repressor element-1 silencing transcription/neuron-restrictive silencer factor is required for neural sodium channel expression during development of Xenopus., Armisén R., J Neurosci. October 1, 2002; 22 (19): 8347-51.

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