| Monarch Ortholog Phenotypes
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Human (2 sources):
Abnormal bleeding,
Menorrhagia
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Mouse (34 sources):
abnormal acute inflammation,
abnormal epididymal fat pad morphology,
abnormal interleukin level,
abnormal kidney morphology,
abnormal renal glomerulus morphology,
abnormal sternum morphology,
cardiac fibrosis,
cardiovascular system phenotype,
decreased angiogenesis,
decreased circulating interleukin-6 level,
decreased liver triglyceride level,
decreased sensitivity to xenobiotic induced morbidity/mortality,
decreased susceptibility to endotoxin shock,
decreased triglyceride level,
enhanced wound healing,
enlarged lymph nodes,
glomerular crescent,
heart inflammation,
hematopoietic system phenotype,
homeostasis/metabolism phenotype,
immune system phenotype,
increased acute inflammation,
increased angiogenesis,
increased anxiety-related response,
increased body temperature,
increased granulocyte number,
increased oxygen consumption,
increased sensitivity to induced morbidity/mortality,
increased susceptibility to Coronaviridae infection,
increased susceptibility to Coronaviridae infection induced morbidity/mortality,
increased thigmotaxis,
liver/biliary system phenotype,
lung hemorrhage,
respiratory system inflammation
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View all ortholog results at Monarch
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Gene expression phenotype annotations where the gene of interest has been
disrupted (manipulated) or is the gene assayed (assayed). Computed annotations are derived from
differential expression analysis from Xenbase processed GEO data with the criteria of a TPM >= 1,
FDR <= 0.05 and an absolute LogFC >= 2.