Papers associated with brain (and cav1)

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	Caveolin 1 is required for axonal outgrowth of motor neurons and affects Xenopus neuromuscular development., Breuer M., Sci Rep. October 5, 2020; 10 (1): 16446.
	Association of A Novel Splice Site Mutation in P/Q-Type Calcium Channels with Childhood Epilepsy and Late-Onset Slowly Progressive Non-Episodic Cerebellar Ataxia., Stendel C., Int J Mol Sci. May 27, 2020; 21 (11):
	Innate Immune Response and Off-Target Mis-splicing Are Common Morpholino-Induced Side Effects in Xenopus., Gentsch GE., Dev Cell. March 12, 2018; 44 (5): 597-610.e10.
	The role of voltage-gated calcium channels in neurotransmitter phenotype specification: Coexpression and functional analysis in Xenopus laevis., Lewis BB., J Comp Neurol. August 1, 2014; 522 (11): 2518-31.
	Age-related homeostatic midchannel proteolysis of neuronal L-type voltage-gated Ca²⁺ channels., Michailidis IE., Neuron. June 4, 2014; 82 (5): 1045-57.
	RAB8B is required for activity and caveolar endocytosis of LRP6., Demir K., Cell Rep. September 26, 2013; 4 (6): 1224-34.
	Molecular cloning and characterization of a hamster Cav1.3 Ca2+ channel variant with a long carboxyl terminus., Kang HW., Biochim Biophys Acta. June 1, 2011; 1808 (6): 1629-38.
	Cloning and characterization of voltage-gated calcium channel alpha1 subunits in Xenopus laevis during development., Lewis BB., Dev Dyn. November 1, 2009; 238 (11): 2891-902.
	Database of queryable gene expression patterns for Xenopus., Gilchrist MJ., Dev Dyn. June 1, 2009; 238 (6): 1379-88.
	BKCa-Cav channel complexes mediate rapid and localized Ca2+-activated K+ signaling., Berkefeld H., Science. October 27, 2006; 314 (5799): 615-20.
	Regulation of maximal open probability is a separable function of Ca(v)beta subunit in L-type Ca2+ channel, dependent on NH2 terminus of alpha1C (Ca(v)1.2alpha)., Kanevsky N., J Gen Physiol. July 1, 2006; 128 (1): 15-36.
	Ca1.2 and CaV1.3 neuronal L-type calcium channels: differential targeting and signaling to pCREB., Zhang H., Eur J Neurosci. May 1, 2006; 23 (9): 2297-310.
	Severe arrhythmia disorder caused by cardiac L-type calcium channel mutations., Splawski I., Proc Natl Acad Sci U S A. June 7, 2005; 102 (23): 8089-96; discussion 8086-8.
	Association of CaV1.3 L-type calcium channels with Shank., Zhang H., J Neurosci. February 2, 2005; 25 (5): 1037-49.

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