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Summary Anatomy Item Literature (7381) Expression Attributions Wiki
XB-ANAT-11

Papers associated with brain (and grin1)

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Postsynaptic and Presynaptic NMDARs Have Distinct Roles in Visual Circuit Development., Kesner P., Cell Rep. January 1, 2020; 32 (4): 107955.                                            


De novo GRIN variants in NMDA receptor M2 channel pore-forming loop are associated with neurological diseases., Li J., Hum Mutat. January 1, 2019; 40 (12): 2393-2413.


An NMDAR positive and negative allosteric modulator series share a binding site and are interconverted by methyl groups., Perszyk R., Elife. January 1, 2018; 7                                                                         


All naturally occurring autoantibodies against the NMDA receptor subunit NR1 have pathogenic potential irrespective of epitope and immunoglobulin class., Castillo-Gómez E., Mol Psychiatry. December 1, 2017; 22 (12): 1776-1784.


A steroid modulatory domain in NR2A collaborates with NR1 exon-5 to control NMDAR modulation by pregnenolone sulfate and protons., Kostakis E., J Neurochem. November 1, 2011; 119 (3): 486-96.


The DREAM protein negatively regulates the NMDA receptor through interaction with the NR1 subunit., Zhang Y, Zhang Y., J Neurosci. June 2, 2010; 30 (22): 7575-86.


Regulation of radial glial motility by visual experience., Tremblay M., J Neurosci. November 11, 2009; 29 (45): 14066-76.                


The serine protease plasmin cleaves the amino-terminal domain of the NR2A subunit to relieve zinc inhibition of the N-methyl-D-aspartate receptors., Yuan H., J Biol Chem. May 8, 2009; 284 (19): 12862-73.


Cloning and Phylogenetic Analysis of NMDA Receptor Subunits NR1, NR2A and NR2B in Xenopus laevis Tadpoles., Ewald RC., Front Mol Neurosci. January 1, 2009; 2 4.          


Differential effect of high pressure on NMDA receptor currents in Xenopus laevis oocytes., Mor A., Diving Hyperb Med. December 1, 2008; 38 (4): 194-6.


Molecular and functional studies of tilapia (Oreochromis mossambicus) NMDA receptor NR1 subunits., Tzeng DW., Comp Biochem Physiol B Biochem Mol Biol. March 1, 2007; 146 (3): 402-11.


Effects of anesthetics on mutant N-methyl-D-aspartate receptors expressed in Xenopus oocytes., Ogata J., J Pharmacol Exp Ther. July 1, 2006; 318 (1): 434-43.


Monoamines directly inhibit N-methyl-D-aspartate receptors expressed in Xenopus oocytes in a voltage-dependent manner., Masuko T., Neurosci Lett. November 16, 2004; 371 (1): 30-3.


Inhibition of the NMDA response by pregnenolone sulphate reveals subtype selective modulation of NMDA receptors by sulphated steroids., Malayev A., Br J Pharmacol. February 1, 2002; 135 (4): 901-9.


Ethanol inhibition of N-methyl-D-aspartate receptors is reduced by site-directed mutagenesis of a transmembrane domain phenylalanine residue., Ronald KM., J Biol Chem. November 30, 2001; 276 (48): 44729-35.


The anti-craving compound acamprosate acts as a weak NMDA-receptor antagonist, but modulates NMDA-receptor subunit expression similar to memantine and MK-801., Rammes G., Neuropharmacology. May 1, 2001; 40 (6): 749-60.


Effects of volatile solvents on recombinant N-methyl-D-aspartate receptors expressed in Xenopus oocytes., Cruz SL., Br J Pharmacol. December 1, 2000; 131 (7): 1303-8.


Molecular determinants of coordinated proton and zinc inhibition of N-methyl-D-aspartate NR1/NR2A receptors., Low CM., Proc Natl Acad Sci U S A. September 26, 2000; 97 (20): 11062-7.


NMDA receptor subunit gene expression in the rat brain: a quantitative analysis of endogenous mRNA levels of NR1Com, NR2A, NR2B, NR2C, NR2D and NR3A., Goebel DJ., Brain Res Mol Brain Res. June 8, 1999; 69 (2): 164-70.


Increased NMDA current and spine density in mice lacking the NMDA receptor subunit NR3A., Das S., Nature. May 28, 1998; 393 (6683): 377-81.


Differentiation of glycine antagonist sites of N-methyl-D-aspartate receptor subtypes. Preferential interaction of CGP 61594 with NR1/2B receptors., Honer M., J Biol Chem. May 1, 1998; 273 (18): 11158-63.


Xenopus oocytes express a unitary glutamate receptor endogenously., Soloviev MM., J Mol Biol. October 17, 1997; 273 (1): 14-8.


Subtype-selective inhibition of N-methyl-D-aspartate receptors by haloperidol., Ilyin VI., Mol Pharmacol. December 1, 1996; 50 (6): 1541-50.


Polyamine spider toxins and mammalian N-methyl-D-aspartate receptors. Structural basis for channel blocking and binding of argiotoxin636., Raditsch M., Eur J Biochem. September 1, 1996; 240 (2): 416-26.


Use of subunit-specific antisense oligodeoxynucleotides to define developmental changes in the properties of N-methyl-D-aspartate receptors., Zhong J., Mol Pharmacol. September 1, 1996; 50 (3): 631-8.


The 5''-untranslated region of the N-methyl-D-aspartate receptor NR2A subunit controls efficiency of translation., Wood MW., J Biol Chem. April 5, 1996; 271 (14): 8115-20.


Developmental and regional expression pattern of a novel NMDA receptor-like subunit (NMDAR-L) in the rodent brain., Sucher NJ., J Neurosci. October 1, 1995; 15 (10): 6509-20.


Control of proton sensitivity of the NMDA receptor by RNA splicing and polyamines., Traynelis SF., Science. May 12, 1995; 268 (5212): 873-6.


Recombinant human NMDA homomeric NR1 receptors expressed in mammalian cells form a high-affinity glycine antagonist binding site., Grimwood S., J Neurochem. February 1, 1995; 64 (2): 525-30.


Spermine potentiation of recombinant N-methyl-D-aspartate receptors is affected by subunit composition., Zhang L., Proc Natl Acad Sci U S A. November 8, 1994; 91 (23): 10883-7.


Subunit-specific potentiation of recombinant N-methyl-D-aspartate receptors by histamine., Williams K., Mol Pharmacol. September 1, 1994; 46 (3): 531-41.


The molecular basis of NMDA receptor subtypes: native receptor diversity is predicted by subunit composition., Buller AL., J Neurosci. September 1, 1994; 14 (9): 5471-84.


Mutational analysis of the glycine-binding site of the NMDA receptor: structural similarity with bacterial amino acid-binding proteins., Kuryatov A., Neuron. June 1, 1994; 12 (6): 1291-300.


Effects of nitroprusside and redox reagents on NMDA receptors expressed in Xenopus oocytes., Omerovic A., Brain Res Mol Brain Res. March 1, 1994; 22 (1-4): 89-96.


Selective antagonism of native and cloned kainate and NMDA receptors by polyamine-containing toxins., Brackley PT., J Pharmacol Exp Ther. September 1, 1993; 266 (3): 1573-80.


Molecular cloning, functional expression, and pharmacological characterization of an N-methyl-D-aspartate receptor subunit from human brain., Planells-Cases R., Proc Natl Acad Sci U S A. June 1, 1993; 90 (11): 5057-61.


Zinc potentiates agonist-induced currents at certain splice variants of the NMDA receptor., Hollmann M., Neuron. May 1, 1993; 10 (5): 943-54.


Molecular characterization of the family of the N-methyl-D-aspartate receptor subunits., Ishii T., J Biol Chem. February 5, 1993; 268 (4): 2836-43.


Cloning of an apparent splice variant of the rat N-methyl-D-aspartate receptor NMDAR1 with altered sensitivity to polyamines and activators of protein kinase C., Durand GM., Proc Natl Acad Sci U S A. October 1, 1992; 89 (19): 9359-63.


Combinatorial RNA splicing alters the surface charge on the NMDA receptor., Anantharam V., FEBS Lett. June 22, 1992; 305 (1): 27-30.


Functional characterization of a heteromeric NMDA receptor channel expressed from cloned cDNAs., Meguro H., Nature. May 7, 1992; 357 (6373): 70-4.

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