Papers associated with muscular system (and kcne1)

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	Functional Characterization of a Spectrum of Novel Romano-Ward Syndrome KCNQ1 Variants., Rinné S., Int J Mol Sci. January 10, 2023; 24 (2):
	Virus-Host Interactions of Enteroviruses and Parvovirus B19 in Myocarditis., Ho HT., Cell Physiol Biochem. November 18, 2021; 55 (6): 679-703.
	Pro-arrhythmogenic Effects of the V141M KCNQ1 Mutation in Short QT Syndrome and Its Potential Therapeutic Targets: Insights from Modeling., Lee HC, Lee HC., J Med Biol Eng. October 1, 2017; 37 (5): 780-789.
	Molecular cloning and functional expression of the K + channel K V 7.1 and the regulatory subunit KCNE1 from equine myocardium., Pedersen PJ., Res Vet Sci. August 1, 2017; 113 79-86.
	Ginseng gintonin activates the human cardiac delayed rectifier K+ channel: involvement of Ca2+/calmodulin binding sites., Choi SH., Mol Cells. September 1, 2014; 37 (9): 656-63.
	Functional assembly of Kv7.1/Kv7.5 channels with emerging properties on vascular muscle physiology., Oliveras A., Arterioscler Thromb Vasc Biol. July 1, 2014; 34 (7): 1522-30.
	Domain-domain interactions determine the gating, permeation, pharmacology, and subunit modulation of the IKs ion channel., Zaydman MA., Elife. March 12, 2014; 3 e03606.
	Intracellular ATP binding is required to activate the slowly activating K+ channel I(Ks)., Li Y., Proc Natl Acad Sci U S A. November 19, 2013; 110 (47): 18922-7.
	Involvement of the eukaryotic initiation factor 6 and kermit2/gipc2 in Xenopus laevis pronephros formation., Tussellino M., Int J Dev Biol. January 1, 2012; 56 (5): 357-62.
	Inhibition of the heterotetrameric K+ channel KCNQ1/KCNE1 by the AMP-activated protein kinase., Alesutan I., Mol Membr Biol. February 1, 2011; 28 (2): 79-89.
	Transmembrane potential of GlyCl-expressing instructor cells induces a neoplastic-like conversion of melanocytes via a serotonergic pathway., Blackiston D., Dis Model Mech. January 1, 2011; 4 (1): 67-85.
	PKC activation and PIP(2) depletion underlie biphasic regulation of IKs by Gq-coupled receptors., Matavel A., J Mol Cell Cardiol. May 1, 2009; 46 (5): 704-12.
	Functional implications of KCNE subunit expression for the Kv7.5 (KCNQ5) channel., Roura-Ferrer M., Cell Physiol Biochem. January 1, 2009; 24 (5-6): 325-34.
	Modulation of potassium channel function confers a hyperproliferative invasive phenotype on embryonic stem cells., Morokuma J., Proc Natl Acad Sci U S A. October 28, 2008; 105 (43): 16608-13.
	Counting membrane-embedded KCNE beta-subunits in functioning K+ channel complexes., Morin TJ., Proc Natl Acad Sci U S A. February 5, 2008; 105 (5): 1478-82.
	Chlorthalidone inhibits the KvLQT1 potassium current in guinea-pig ventricular myocytes and oocytes from Xenopus laevis., Mancilla-Simbro C., Br J Pharmacol. February 1, 2008; 153 (3): 448-58.
	Upregulation of KCNE1 induces QT interval prolongation in patients with chronic heart failure., Watanabe E., Circ J. April 1, 2007; 71 (4): 471-8.
	KCNE2 is colocalized with KCNQ1 and KCNE1 in cardiac myocytes and may function as a negative modulator of I(Ks) current amplitude in the heart., Wu DM., Heart Rhythm. December 1, 2006; 3 (12): 1469-80.
	Ancillary subunits and stimulation frequency determine the potency of chromanol 293B block of the KCNQ1 potassium channel., Bett GC., J Physiol. November 1, 2006; 576 (Pt 3): 755-67.
	Frequency-dependent modulation of KCNQ1 and HERG1 potassium channels., Diness TG., Biochem Biophys Res Commun. May 19, 2006; 343 (4): 1224-33.
	De novo KCNQ1 mutation responsible for atrial fibrillation and short QT syndrome in utero., Hong K., Cardiovasc Res. December 1, 2005; 68 (3): 433-40.
	The MinK-related peptides., McCrossan ZA., Neuropharmacology. November 1, 2004; 47 (6): 787-821.
	Ranolazine: ion-channel-blocking actions and in vivo electrophysiological effects., Schram G., Br J Pharmacol. August 1, 2004; 142 (8): 1300-8.
	Association of the serum and glucocorticoid regulated kinase (sgk1) gene with QT interval., Busjahn A., Cell Physiol Biochem. January 1, 2004; 14 (3): 135-42.
	Novel gene hKCNE4 slows the activation of the KCNQ1 channel., Teng S., Biochem Biophys Res Commun. April 11, 2003; 303 (3): 808-13.
	Regulation of KCNE1-dependent K(+) current by the serum and glucocorticoid-inducible kinase (SGK) isoforms., Embark HM., Pflugers Arch. February 1, 2003; 445 (5): 601-6.
	Inhibition of cardiac potassium currents by pentobarbital., Bachmann A., Naunyn Schmiedebergs Arch Pharmacol. January 1, 2002; 365 (1): 29-37.
	Chromanol 293B, a blocker of the slow delayed rectifier K+ current (IKs), inhibits the CFTR Cl- current., Bachmann A., Naunyn Schmiedebergs Arch Pharmacol. June 1, 2001; 363 (6): 590-6.
	minK-related peptide 1 associates with Kv4.2 and modulates its gating function: potential role as beta subunit of cardiac transient outward channel?, Zhang M., Circ Res. May 25, 2001; 88 (10): 1012-9.
	Short- and long-term effects of amiodarone on the two components of cardiac delayed rectifier K(+) current., Kamiya K., Circulation. March 6, 2001; 103 (9): 1317-24.
	MiRP2 forms potassium channels in skeletal muscle with Kv3.4 and is associated with periodic paralysis., Abbott GW., Cell. January 26, 2001; 104 (2): 217-31.
	Inhibition of IKs channels by HMR 1556., Gögelein H., Naunyn Schmiedebergs Arch Pharmacol. December 1, 2000; 362 (6): 480-8.
	Norpropoxyphene-induced cardiotoxicity is associated with changes in ion-selectivity and gating of HERG currents., Ulens C., Cardiovasc Res. December 1, 1999; 44 (3): 568-78.
	Single-channel characteristics of wild-type IKs channels and channels formed with two minK mutants that cause long QT syndrome., Sesti F., J Gen Physiol. December 1, 1998; 112 (6): 651-63.
	Independent and exclusive modulation of cardiac delayed rectifying K+ current by protein kinase C and protein kinase A., Lo CF., Circ Res. November 16, 1998; 83 (10): 995-1002.
	Activation and inactivation of homomeric KvLQT1 potassium channels., Pusch M., Biophys J. August 1, 1998; 75 (2): 785-92.
	A novel benzodiazepine that activates cardiac slow delayed rectifier K+ currents., Salata JJ., Mol Pharmacol. July 1, 1998; 54 (1): 220-30.
	Effects of the chromanol 293B, a selective blocker of the slow, component of the delayed rectifier K+ current, on repolarization in human and guinea pig ventricular myocytes., Bosch RF., Cardiovasc Res. May 1, 1998; 38 (2): 441-50.
	Blockade of HERG channels by the class III antiarrhythmic azimilide: mode of action., Busch AE., Br J Pharmacol. January 1, 1998; 123 (1): 23-30.
	Suppression of slow delayed rectifier current by a truncated isoform of KvLQT1 cloned from normal human heart., Jiang M., J Biol Chem. September 26, 1997; 272 (39): 24109-12.
	KVLQT channels are inhibited by the K+ channel blocker 293B., Bleich M., Pflugers Arch. August 1, 1997; 434 (4): 499-501.
	A minK-HERG complex regulates the cardiac potassium current I(Kr)., McDonald TV., Nature. July 17, 1997; 388 (6639): 289-92.
	Mechanism of enhancement of slow delayed rectifier current by extracellular sulfhydryl modification., Yao JA., Am J Physiol. July 1, 1997; 273 (1 Pt 2): H208-19.
	I(sK) Channel in Strial Marginal Cells. Voltage-Dependence, Ion-Selectivity, Inhibition by 293B and Sensitivity to Clofilium., Shen Z., Audit Neurosci. January 1, 1997; 3 (3): 215-230.
	IK of rabbit ventricle is composed of two currents: evidence for IKs., Salata JJ., Am J Physiol. December 1, 1996; 271 (6 Pt 2): H2477-89.
	Coassembly of K(V)LQT1 and minK (IsK) proteins to form cardiac I(Ks) potassium channel., Sanguinetti MC., Nature. November 7, 1996; 384 (6604): 80-3.
	Inhibition of IKs in guinea pig cardiac myocytes and guinea pig IsK channels by the chromanol 293B., Busch AE., Pflugers Arch. October 1, 1996; 432 (6): 1094-6.
	A corticosteroid-induced gene expressing an "IsK-like" K+ channel activity in Xenopus oocytes., Attali B., Proc Natl Acad Sci U S A. June 20, 1995; 92 (13): 6092-6.
	The novel class III antiarrhythmics NE-10064 and NE-10133 inhibit IsK channels expressed in Xenopus oocytes and IKs in guinea pig cardiac myocytes., Busch AE., Biochem Biophys Res Commun. July 15, 1994; 202 (1): 265-70.
	K+ currents expressed from the guinea pig cardiac IsK protein are enhanced by activators of protein kinase C., Zhang ZJ., Proc Natl Acad Sci U S A. March 1, 1994; 91 (5): 1766-70.

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