Papers associated with regenerating tissue (and kcne1)

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	Polyunsaturated fatty acid analogs act antiarrhythmically on the cardiac IKs channel., Liin SI., Proc Natl Acad Sci U S A. May 5, 2015; 112 (18): 5714-9.
	High incidence of functional ion-channel abnormalities in a consecutive Long QT cohort with novel missense genetic variants of unknown significance., Steffensen AB., Sci Rep. January 12, 2015; 5 10009.
	Ginseng gintonin activates the human cardiac delayed rectifier K+ channel: involvement of Ca2+/calmodulin binding sites., Choi SH., Mol Cells. September 1, 2014; 37 (9): 656-63.
	Domain-domain interactions determine the gating, permeation, pharmacology, and subunit modulation of the IKs ion channel., Zaydman MA., Elife. March 12, 2014; 3 e03606.
	Differential effects of ginsenoside metabolites on slowly activating delayed rectifier K(+) and KCNQ1 K(+) channel currents., Choi SH., J Ginseng Res. July 1, 2013; 37 (3): 324-31.
	Mink1 regulates β-catenin-independent Wnt signaling via Prickle phosphorylation., Daulat AM., Mol Cell Biol. January 1, 2012; 32 (1): 173-85.
	Involvement of the eukaryotic initiation factor 6 and kermit2/gipc2 in Xenopus laevis pronephros formation., Tussellino M., Int J Dev Biol. January 1, 2012; 56 (5): 357-62.
	KCNQ1 subdomains involved in KCNE modulation revealed by an invertebrate KCNQ1 orthologue., Nakajo K., J Gen Physiol. November 1, 2011; 138 (5): 521-35.
	Extracellular potassium inhibits Kv7.1 potassium channels by stabilizing an inactivated state., Larsen AP., Biophys J. August 17, 2011; 101 (4): 818-27.
	Transmembrane potential of GlyCl-expressing instructor cells induces a neoplastic-like conversion of melanocytes via a serotonergic pathway., Blackiston D., Dis Model Mech. January 1, 2011; 4 (1): 67-85.
	KCNE1 and KCNE3 beta-subunits regulate membrane surface expression of Kv12.2 K(+) channels in vitro and form a tripartite complex in vivo., Clancy SM., PLoS One. July 22, 2009; 4 (7): e6330.
	Discovery of a novel activator of KCNQ1-KCNE1 K channel complexes., Mruk K., PLoS One. January 1, 2009; 4 (1): e4236.
	Modulation of potassium channel function confers a hyperproliferative invasive phenotype on embryonic stem cells., Morokuma J., Proc Natl Acad Sci U S A. October 28, 2008; 105 (43): 16608-13.
	KCNE1 constrains the voltage sensor of Kv7.1 K+ channels., Shamgar L., PLoS One. April 9, 2008; 3 (4): e1943.
	KCNE peptides differently affect voltage sensor equilibrium and equilibration rates in KCNQ1 K+ channels., Rocheleau JM., J Gen Physiol. January 1, 2008; 131 (1): 59-68.
	The role of S4 charges in voltage-dependent and voltage-independent KCNQ1 potassium channel complexes., Panaghie G., J Gen Physiol. February 1, 2007; 129 (2): 121-33.
	Secondary structure of a KCNE cytoplasmic domain., Rocheleau JM., J Gen Physiol. December 1, 2006; 128 (6): 721-9.
	KCNE3 truncation mutants reveal a bipartite modulation of KCNQ1 K+ channels., Gage SD., J Gen Physiol. December 1, 2004; 124 (6): 759-71.
	Acute effects of dronedarone on both components of the cardiac delayed rectifier K+ current, HERG and KvLQT1/minK potassium channels., Thomas D., Br J Pharmacol. November 1, 2003; 140 (5): 996-1002.
	KCNE2 modulates current amplitudes and activation kinetics of HCN4: influence of KCNE family members on HCN4 currents., Decher N., Pflugers Arch. September 1, 2003; 446 (6): 633-40.
	Clinical and electrophysiological characterization of a novel mutation (F193L) in the KCNQ1 gene associated with long QT syndrome., Yamaguchi M., Clin Sci (Lond). April 1, 2003; 104 (4): 377-82.
	Inhibitory effects of volatile anesthetics on currents produced on heterologous expression of KvLQT1 and minK in Xenopus oocytes., Chen X., Vascul Pharmacol. July 1, 2002; 39 (1-2): 33-8.
	The oxidant thimerosal modulates gating behavior of KCNQ1 by interaction with the channel outer shell., Kerst G., J Membr Biol. March 15, 2002; 186 (2): 89-100.
	Blocking effects of the antiarrhythmic drug propafenone on the HERG potassium channel., Mergenthaler J., Naunyn Schmiedebergs Arch Pharmacol. April 1, 2001; 363 (4): 472-80.
	MinK subdomains that mediate modulation of and association with KvLQT1., Tapper AR., J Gen Physiol. September 1, 2000; 116 (3): 379-90.
	Gating and flickery block differentially affected by rubidium in homomeric KCNQ1 and heteromeric KCNQ1/KCNE1 potassium channels., Pusch M., Biophys J. January 1, 2000; 78 (1): 211-26.
	Single-channel characteristics of wild-type IKs channels and channels formed with two minK mutants that cause long QT syndrome., Sesti F., J Gen Physiol. December 1, 1998; 112 (6): 651-63.
	Increase of the single-channel conductance of KvLQT1 potassium channels induced by the association with minK., Pusch M., Pflugers Arch. December 1, 1998; 437 (1): 172-4.
	Activation and inactivation of homomeric KvLQT1 potassium channels., Pusch M., Biophys J. August 1, 1998; 75 (2): 785-92.
	Voltage-dependent inactivation of the human K+ channel KvLQT1 is eliminated by association with minimal K+ channel (minK) subunits., Tristani-Firouzi M., J Physiol. July 1, 1998; 510 ( Pt 1) 37-45.
	Blockade of HERG channels by the class III antiarrhythmic azimilide: mode of action., Busch AE., Br J Pharmacol. January 1, 1998; 123 (1): 23-30.
	Block by propofol and thiopentone of the min K current (IsK) expressed in Xenopus oocytes., Heath BM., Naunyn Schmiedebergs Arch Pharmacol. September 1, 1997; 356 (3): 404-9.
	Species variants of the IsK protein: differences in kinetics, voltage dependence, and La3+ block of the currents expressed in Xenopus oocytes., Hice RE., Pflugers Arch. January 1, 1994; 426 (1-2): 139-45.

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