Papers associated with oocyte (and slc2a1)

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	Development of a rapid functional assay that predicts GLUT1 disease severity., Zaman SM., Neurol Genet. December 6, 2018; 4 (6): e297.
	Human Mutations in SLC2A9 (Glut9) Affect Transport Capacity for Urate., Ruiz A., Front Physiol. January 1, 2018; 9 476.
	New fluorinated fructose analogs as selective probes of the hexose transporter protein GLUT5., Soueidan OM., Org Biomol Chem. June 21, 2015; 13 (23): 6511-21.
	Expression, purification, and structural insights for the human uric acid transporter, GLUT9, using the Xenopus laevis oocytes system., Clémençon B., PLoS One. January 1, 2014; 9 (10): e108852.
	FGT-1 is the major glucose transporter in C. elegans and is central to aging pathways., Feng Y., Biochem J. December 1, 2013; 456 (2): 219-29.
	The human sodium-dependent ascorbic acid transporters SLC23A1 and SLC23A2 do not mediate ascorbic acid release in the proximal renal epithelial cell., Eck P., Physiol Rep. November 1, 2013; 1 (6): e00136.
	Intestinal dehydroascorbic acid (DHA) transport mediated by the facilitative sugar transporters, GLUT2 and GLUT8., Corpe CP., J Biol Chem. March 29, 2013; 288 (13): 9092-101.
	Characterization of bovine glucose transporter 1 kinetics and substrate specificities in Xenopus oocytes., Bentley PA., J Dairy Sci. March 1, 2012; 95 (3): 1188-97.
	Ligand-induced movements of inner transmembrane helices of Glut1 revealed by chemical cross-linking of di-cysteine mutants., Mueckler M., PLoS One. January 1, 2012; 7 (2): e31412.
	Functional studies of the T295M mutation causing Glut1 deficiency: glucose efflux preferentially affected by T295M., Wang D., Pediatr Res. November 1, 2008; 64 (5): 538-43.
	SLC2A9 is a high-capacity urate transporter in humans., Caulfield MJ., PLoS Med. October 7, 2008; 5 (10): e197.
	Paroxysmal exercise-induced dyskinesia and epilepsy is due to mutations in SLC2A1, encoding the glucose transporter GLUT1., Suls A., Brain. July 1, 2008; 131 (Pt 7): 1831-44.
	6-Bromo-6-deoxy-L-ascorbic acid: an ascorbate analog specific for Na+-dependent vitamin C transporter but not glucose transporter pathways., Corpe CP., J Biol Chem. February 18, 2005; 280 (7): 5211-20.
	A structural basis for the acute effects of HIV protease inhibitors on GLUT4 intrinsic activity., Hertel J., J Biol Chem. December 31, 2004; 279 (53): 55147-52.
	Indinavir inhibits the glucose transporter isoform Glut4 at physiologic concentrations., Murata H., AIDS. April 12, 2002; 16 (6): 859-63.
	Dehydroascorbic acid transport by GLUT4 in Xenopus oocytes and isolated rat adipocytes., Rumsey SC., J Biol Chem. September 8, 2000; 275 (36): 28246-53.
	Cysteine-scanning mutagenesis of transmembrane segment 11 of the GLUT1 facilitative glucose transporter., Hruz PW., Biochemistry. August 8, 2000; 39 (31): 9367-72.
	Cysteine-scanning mutagenesis of transmembrane segment 7 of the GLUT1 glucose transporter., Hruz PW., J Biol Chem. December 17, 1999; 274 (51): 36176-80.
	Glucose transporter isoforms GLUT1 and GLUT3 transport dehydroascorbic acid., Rumsey SC., J Biol Chem. July 25, 1997; 272 (30): 18982-9.
	Insulin and insulin-like growth factor I (IGF-I) stimulate GLUT4 glucose transporter translocation in Xenopus oocytes., Mora S., Biochem J. October 1, 1995; 311 ( Pt 1) 59-65.
	Role of the C-terminal tail of the GLUT1 glucose transporter in its expression and function in Xenopus laevis oocytes., Due AD., Biochemistry. April 25, 1995; 34 (16): 5462-71.
	Coupling of glucose transport and phosphorylation in Xenopus oocytes and cultured cells: determination of the rate-limiting step., Whitesell RR., J Cell Physiol. December 1, 1993; 157 (3): 509-18.
	GTP analogs suppress uptake but not transport of D-glucose analogs in Glut1 glucose transporter-expressing Xenopus oocytes., Wellner M., FEBS Lett. July 19, 1993; 327 (1): 95-8.
	Evidence from oocyte expression that the erythrocyte water channel is distinct from band 3 and the glucose transporter., Zhang R., J Clin Invest. November 1, 1991; 88 (5): 1553-8.

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