Papers associated with oocyte (and kcne1)

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	Nocturnal Atrial Fibrillation Caused by Mutation in KCND2, Encoding Pore-Forming (α) Subunit of the Cardiac Kv4.2 Potassium Channel., Drabkin M., Circ Genom Precis Med. November 1, 2018; 11 (11): e002293.
	KCNE1 tunes the sensitivity of KV7.1 to polyunsaturated fatty acids by moving turret residues close to the binding site., Larsson JE., Elife. July 17, 2018; 7
	Direct neurotransmitter activation of voltage-gated potassium channels., Manville RW., Nat Commun. May 10, 2018; 9 (1): 1847.
	Insulin treatment augments KCNQ1/KCNE1 currents but not KCNQ1 currents, which is associated with an increase in KCNE1 expression., Wu M., Biochem Biophys Res Commun. November 4, 2017; 493 (1): 409-415.
	KCNE1 induces fenestration in the Kv7.1/KCNE1 channel complex that allows for highly specific pharmacological targeting., Wrobel E., Nat Commun. October 12, 2016; 7 12795.
	Ginseng gintonin activates the human cardiac delayed rectifier K+ channel: involvement of Ca2+/calmodulin binding sites., Choi SH., Mol Cells. September 1, 2014; 37 (9): 656-63.
	Functional assembly of Kv7.1/Kv7.5 channels with emerging properties on vascular muscle physiology., Oliveras A., Arterioscler Thromb Vasc Biol. July 1, 2014; 34 (7): 1522-30.
	Domain-domain interactions determine the gating, permeation, pharmacology, and subunit modulation of the IKs ion channel., Zaydman MA., Elife. March 12, 2014; 3 e03606.
	Impaired ion channel function related to a common KCNQ1 mutation - implications for risk stratification in long QT syndrome 1., Aidery P., Gene. December 10, 2012; 511 (1): 26-33.
	AMP-activated protein kinase in BK-channel regulation and protection against hearing loss following acoustic overstimulation., Föller M., FASEB J. October 1, 2012; 26 (10): 4243-53.
	Inhibition of cardiac Kir2.1-2.3 channels by beta3 adrenoreceptor antagonist SR 59230A., Kulzer M., Biochem Biophys Res Commun. July 27, 2012; 424 (2): 315-20.
	KCNQ1 subdomains involved in KCNE modulation revealed by an invertebrate KCNQ1 orthologue., Nakajo K., J Gen Physiol. November 1, 2011; 138 (5): 521-35.
	Biophysical properties of mutant KCNQ1 S277L channels linked to hereditary long QT syndrome with phenotypic variability., Aidery P., Biochim Biophys Acta. April 1, 2011; 1812 (4): 488-94.
	Functional delivery of a membrane protein into oocyte membranes using bicelles., Kang C., Biochemistry. February 2, 2010; 49 (4): 653-5.
	The G314S KCNQ1 mutation exerts a dominant-negative effect on expression of KCNQ1 channels in oocytes., Li W., Biochem Biophys Res Commun. May 29, 2009; 383 (2): 206-9.
	Modulation of potassium channel function confers a hyperproliferative invasive phenotype on embryonic stem cells., Morokuma J., Proc Natl Acad Sci U S A. October 28, 2008; 105 (43): 16608-13.
	KCNQ1 and KCNE1 in the IKs channel complex make state-dependent contacts in their extracellular domains., Xu X., J Gen Physiol. June 1, 2008; 131 (6): 589-603.
	The KCNE1 beta-subunit exerts a transient effect on the KCNQ1 K+ channel., Poulsen AN., Biochem Biophys Res Commun. November 9, 2007; 363 (1): 133-9.
	KCNE1 and KCNE3 stabilize and/or slow voltage sensing S4 segment of KCNQ1 channel., Nakajo K., J Gen Physiol. September 1, 2007; 130 (3): 269-81.
	Secondary structure of a KCNE cytoplasmic domain., Rocheleau JM., J Gen Physiol. December 1, 2006; 128 (6): 721-9.
	Endogenous KCNE subunits govern Kv2.1 K+ channel activation kinetics in Xenopus oocyte studies., Gordon E., Biophys J. February 15, 2006; 90 (4): 1223-31.
	KCNE3 truncation mutants reveal a bipartite modulation of KCNQ1 K+ channels., Gage SD., J Gen Physiol. December 1, 2004; 124 (6): 759-71.
	Identification and characterisation of a novel KCNQ1 mutation in a family with Romano-Ward syndrome., Zehelein J., Biochim Biophys Acta. November 5, 2004; 1690 (3): 185-92.
	Automated higher-throughput compound screening on ion channel targets based on the Xenopus laevis oocyte expression system., Pehl U., Assay Drug Dev Technol. October 1, 2004; 2 (5): 515-24.
	Characterization of a novel Long QT syndrome mutation G52R-KCNE1 in a Chinese family., Ma L., Cardiovasc Res. September 1, 2003; 59 (3): 612-9.
	RNA interference reveals that endogenous Xenopus MinK-related peptides govern mammalian K+ channel function in oocyte expression studies., Anantharam A., J Biol Chem. April 4, 2003; 278 (14): 11739-45.
	minK-related peptide 1 associates with Kv4.2 and modulates its gating function: potential role as beta subunit of cardiac transient outward channel?, Zhang M., Circ Res. May 25, 2001; 88 (10): 1012-9.
	MinK subdomains that mediate modulation of and association with KvLQT1., Tapper AR., J Gen Physiol. September 1, 2000; 116 (3): 379-90.
	Long QT syndrome-associated mutations in the S4-S5 linker of KvLQT1 potassium channels modify gating and interaction with minK subunits., Franqueza L., J Biol Chem. July 23, 1999; 274 (30): 21063-70.
	Single-channel properties of IKs potassium channels., Yang Y., J Gen Physiol. December 1, 1998; 112 (6): 665-78.
	Single-channel characteristics of wild-type IKs channels and channels formed with two minK mutants that cause long QT syndrome., Sesti F., J Gen Physiol. December 1, 1998; 112 (6): 651-63.
	Activation and inactivation of homomeric KvLQT1 potassium channels., Pusch M., Biophys J. August 1, 1998; 75 (2): 785-92.
	Suppression of slow delayed rectifier current by a truncated isoform of KvLQT1 cloned from normal human heart., Jiang M., J Biol Chem. September 26, 1997; 272 (39): 24109-12.
	KVLQT channels are inhibited by the K+ channel blocker 293B., Bleich M., Pflugers Arch. August 1, 1997; 434 (4): 499-501.
	Mechanism of enhancement of slow delayed rectifier current by extracellular sulfhydryl modification., Yao JA., Am J Physiol. July 1, 1997; 273 (1 Pt 2): H208-19.
	cAMP increases apical IsK channel current and K+ secretion in vestibular dark cells., Sunose H., J Membr Biol. March 1, 1997; 156 (1): 25-35.
	MinK potassium channels are heteromultimeric complexes., Tai KK., J Biol Chem. January 17, 1997; 272 (3): 1654-8.
	A corticosteroid-induced gene expressing an "IsK-like" K+ channel activity in Xenopus oocytes., Attali B., Proc Natl Acad Sci U S A. June 20, 1995; 92 (13): 6092-6.
	The minK potassium channel exists in functional and nonfunctional forms when expressed in the plasma membrane of Xenopus oocytes., Blumenthal EM., J Neurosci. May 1, 1994; 14 (5 Pt 2): 3097-105.
	Time dependent changes in biophysical properties of minK channels expressed in Xenopus oocytes., Busch AE., Biochem Biophys Res Commun. December 15, 1993; 197 (2): 473-7.
	Are Xenopus oocytes unique in displaying functional IsK channel heterologous expression?, Lesage F., Recept Channels. January 1, 1993; 1 (2): 143-52.
	Modulation by cAMP of a slowly activating potassium channel expressed in Xenopus oocytes., Blumenthal EM., J Neurosci. January 1, 1992; 12 (1): 290-6.
	Alteration of channel activities and gating by mutations of slow ISK potassium channel., Takumi T., J Biol Chem. November 25, 1991; 266 (33): 22192-8.

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