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APC/C-mediated multiple monoubiquitylation provides an alternative degradation signal for cyclin B1. , Dimova NV., Nat Cell Biol. January 29, 2012; 14 (2): 168-76.
Restraint of apoptosis during mitosis through interdomain phosphorylation of caspase-2. , Andersen JL., EMBO J. October 21, 2009; 28 (20): 3216-27.
Phosphorylation of the cyclin b1 cytoplasmic retention sequence by mitogen-activated protein kinase and Plx. , Walsh S., Mol Cancer Res. February 1, 2003; 1 (4): 280-9.
The APC regulator CDH1 is essential for the progression of embryonic cell cycles in Xenopus. , Zhou Y., Biochem Biophys Res Commun. May 31, 2002; 294 (1): 120-6.
Two distinct mechanisms control the accumulation of cyclin B1 and Mos in Xenopus oocytes in response to progesterone. , Frank-Vaillant M., Mol Biol Cell. October 1, 1999; 10 (10): 3279-88.
The mitogen-activated protein kinase signaling pathway stimulates mos mRNA cytoplasmic polyadenylation during Xenopus oocyte maturation. , Howard EL., Mol Cell Biol. March 1, 1999; 19 (3): 1990-9.
Maternal Xenopus Cdk2-cyclin E complexes function during meiotic and early embryonic cell cycles that lack a G1 phase. , Rempel RE., J Biol Chem. March 24, 1995; 270 (12): 6843-55.
Mitotic repression of RNA polymerase III transcription in vitro mediated by phosphorylation of a TFIIIB component. , Gottesfeld JM., Science. January 7, 1994; 263 (5143): 81-4.
Requirement of mosXe protein kinase for meiotic maturation of Xenopus oocytes induced by a cdc2 mutant lacking regulatory phosphorylation sites. , Pickham KM., Mol Cell Biol. July 1, 1992; 12 (7): 3192-203.
On the synthesis and destruction of A- and B-type cyclins during oogenesis and meiotic maturation in Xenopus laevis. , Kobayashi H., J Cell Biol. August 1, 1991; 114 (4): 755-65.
Phosphorylation of Xenopus cyclins B1 and B2 is not required for cell cycle transitions. , Izumi T., Mol Cell Biol. August 1, 1991; 11 (8): 3860-7.
Meiotic induction by Xenopus cyclin B is accelerated by coexpression with mosXe. , Freeman RS., Mol Cell Biol. March 1, 1991; 11 (3): 1713-7.