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Binding of sulphonylureas to plasma proteins - A KATP channel perspective. , Proks P., PLoS One. May 15, 2018; 13 (5): e0197634.
Xenopus as a model system for studying pancreatic development and diabetes. , Kofent J., Semin Cell Dev Biol. March 1, 2016; 51 106-16.
Recessive mutations in PCBD1 cause a new type of early-onset diabetes. , Simaite D., Diabetes. October 1, 2014; 63 (10): 3557-64.
Homeoprotein hhex-induced conversion of intestinal to ventral pancreatic precursors results in the formation of giant pancreata in Xenopus embryos. , Zhao H ., Proc Natl Acad Sci U S A. May 29, 2012; 109 (22): 8594-9.
Transient expression of Ngn3 in Xenopus endoderm promotes early and ectopic development of pancreatic beta and delta cells. , Oropeza D., Genesis. March 1, 2012; 50 (3): 271-85.
Neurally Derived Tissues in Xenopus laevis Embryos Exhibit a Consistent Bioelectrical Left- Right Asymmetry. , Pai VP ., Stem Cells Int. January 1, 2012; 2012 353491.
The ATP-sensitive K(+)-channel (K(ATP)) controls early left- right patterning in Xenopus and chick embryos. , Aw S., Dev Biol. October 1, 2010; 346 (1): 39-53.
Xenopus pancreas development. , Pearl EJ ., Dev Dyn. June 1, 2009; 238 (6): 1271-86.
Expression of complement components coincides with early patterning and organogenesis in Xenopus laevis. , McLin VA ., Int J Dev Biol. January 1, 2008; 52 (8): 1123-33.
A heterozygous activating mutation in the sulphonylurea receptor SUR1 ( ABCC8) causes neonatal diabetes. , Proks P., Hum Mol Genet. June 1, 2006; 15 (11): 1793-800.
Functional effects of naturally occurring KCNJ11 mutations causing neonatal diabetes on cloned cardiac KATP channels. , Tammaro P., J Physiol. February 15, 2006; 571 (Pt 1): 3-14.
Open state destabilization by ATP occupancy is mechanism speeding burst exit underlying KATP channel inhibition by ATP. , Li L., J Gen Physiol. January 1, 2002; 119 (1): 105-16.
Amiloride derivatives are potent blockers of KATP channels. , Bollensdorff C., Naunyn Schmiedebergs Arch Pharmacol. October 1, 2001; 364 (4): 351-8.
Chromanol 293B, a blocker of the slow delayed rectifier K+ current (IKs), inhibits the CFTR Cl- current. , Bachmann A., Naunyn Schmiedebergs Arch Pharmacol. June 1, 2001; 363 (6): 590-6.
Glimepiride block of cloned beta-cell, cardiac and smooth muscle K(ATP) channels. , Song DK., Br J Pharmacol. May 1, 2001; 133 (1): 193-9.
Effects of mitiglinide (S 21403) on Kir6.2/ SUR1, Kir6.2/ SUR2A and Kir6.2/SUR2B types of ATP-sensitive potassium channel. , Reimann F., Br J Pharmacol. April 1, 2001; 132 (7): 1542-8.
Nucleotide modulation of pinacidil stimulation of the cloned K(ATP) channel Kir6.2/ SUR2A. , Gribble FM., Mol Pharmacol. June 1, 2000; 57 (6): 1256-61.
Rat homolog of sulfonylurea receptor 2B determines glibenclamide sensitivity of ROMK2 in Xenopus laevis oocyte. , Tanemoto M., Am J Physiol Renal Physiol. April 1, 2000; 278 (4): F659-66.
Altered functional properties of KATP channel conferred by a novel splice variant of SUR1. , Sakura H., J Physiol. December 1, 1999; 521 Pt 2 337-50.
Pharmacological plasticity of cardiac ATP-sensitive potassium channels toward diazoxide revealed by ADP. , D'hahan N., Proc Natl Acad Sci U S A. October 12, 1999; 96 (21): 12162-7.
Differential sensitivity of beta-cell and extrapancreatic K(ATP) channels to gliclazide. , Gribble FM., Diabetologia. July 1, 1999; 42 (7): 845-8.
Identification of the high-affinity tolbutamide site on the SUR1 subunit of the K(ATP) channel. , Ashfield R., Diabetes. June 1, 1999; 48 (6): 1341-7.
Tissue specificity of sulfonylureas: studies on cloned cardiac and beta-cell K(ATP) channels. , Gribble FM., Diabetes. September 1, 1998; 47 (9): 1412-8.
Cloning and characterization of a novel human inwardly rectifying potassium channel predominantly expressed in small intestine. , Partiseti M., FEBS Lett. August 28, 1998; 434 (1-2): 171-6.
Phentolamine block of KATP channels is mediated by Kir6.2. , Proks P., Proc Natl Acad Sci U S A. October 14, 1997; 94 (21): 11716-20.
Expression of Xenopus snail in mesoderm and prospective neural fold ectoderm. , Essex LJ., Dev Dyn. October 1, 1993; 198 (2): 108-22.