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Summary Anatomy Item Literature (8703) Expression Attributions Wiki
XB-ANAT-506

Papers associated with embryonic structure (and gli2)

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Prdm15 acts upstream of Wnt4 signaling in anterior neural development of Xenopus laevis., Saumweber E., Front Cell Dev Biol. January 1, 2024; 12 1316048.                            


Cilia-localized GID/CTLH ubiquitin ligase complex regulates protein homeostasis of sonic hedgehog signaling components., Hantel F., J Cell Sci. May 1, 2022; 135 (9):                                     


Endosome-Mediated Epithelial Remodeling Downstream of Hedgehog-Gli Is Required for Tracheoesophageal Separation., Nasr T., Dev Cell. December 16, 2019; 51 (6): 665-674.e6.                  


Xenopus slc7a5 is essential for notochord function and eye development., Katada T., Mech Dev. February 1, 2019; 155 48-59.                


Gli2 is required for the induction and migration of Xenopus laevis neural crest., Cerrizuela S., Mech Dev. December 1, 2018; 154 219-239.                      


Members of the Rusc protein family interact with Sufu and inhibit vertebrate Hedgehog signaling., Jin Z., Development. November 1, 2016; 143 (21): 3944-3955.                        


A Retinoic Acid-Hedgehog Cascade Coordinates Mesoderm-Inducing Signals and Endoderm Competence during Lung Specification., Rankin SA, Rankin SA., Cell Rep. June 28, 2016; 16 (1): 66-78.                                              


Prepatterning and patterning of the thalamus along embryonic development of Xenopus laevis., Bandín S., Front Neuroanat. February 3, 2015; 9 107.                                                    


Chibby functions in Xenopus ciliary assembly, embryonic development, and the regulation of gene expression., Shi J., Dev Biol. November 15, 2014; 395 (2): 287-98.                    


Gene regulatory networks governing lung specification., Rankin SA, Rankin SA., J Cell Biochem. August 1, 2014; 115 (8): 1343-50.


Stabilization of speckle-type POZ protein (Spop) by Daz interacting protein 1 (Dzip1) is essential for Gli turnover and the proper output of Hedgehog signaling., Schwend T., J Biol Chem. November 8, 2013; 288 (45): 32809-32820.                


The cytoskeletal protein Zyxin inhibits Shh signaling during the CNS patterning in Xenopus laevis through interaction with the transcription factor Gli1., Martynova NY., Dev Biol. August 1, 2013; 380 (1): 37-48.                      


Indian hedgehog signaling is required for proper formation, maintenance and migration of Xenopus neural crest., Agüero TH., Dev Biol. April 15, 2012; 364 (2): 99-113.                    


Suppressing Wnt signaling by the hedgehog pathway through sFRP-1., He J., J Biol Chem. November 24, 2006; 281 (47): 35598-602.


Cooperative requirement of the Gli proteins in neurogenesis., Nguyen V., Development. July 1, 2005; 132 (14): 3267-79.                      


Loss-of-function mutations in the human GLI2 gene are associated with pituitary anomalies and holoprosencephaly-like features., Roessler E., Proc Natl Acad Sci U S A. November 11, 2003; 100 (23): 13424-9.          


The amino-terminal region of Gli3 antagonizes the Shh response and acts in dorsoventral fate specification in the developing spinal cord., Meyer NP., Dev Biol. May 15, 2003; 257 (2): 343-55.


A novel function for Hedgehog signalling in retinal pigment epithelium differentiation., Perron M., Development. April 1, 2003; 130 (8): 1565-77.                                  


Dorsal-ventral patterning of the spinal cord requires Gli3 transcriptional repressor activity., Persson M., Genes Dev. November 15, 2002; 16 (22): 2865-78.


Conserved expression control and shared activity between cognate T-box genes Tbx2 and Tbx3 in connection with Sonic hedgehog signaling during Xenopus eye development., Takabatake Y., Dev Growth Differ. August 1, 2002; 44 (4): 257-71.              


Anorectal malformations caused by defects in sonic hedgehog signaling., Mo R., Am J Pathol. August 1, 2001; 159 (2): 765-74.


Distinct expression of two types of Xenopus Patched genes during early embryogenesis and hindlimb development., Takabatake T., Mech Dev. November 1, 2000; 98 (1-2): 99-104.            


Gli2 functions in FGF signaling during antero-posterior patterning., Brewster R., Development. October 1, 2000; 127 (20): 4395-405.            


Shh and Wnt signaling pathways converge to control Gli gene activation in avian somites., Borycki A., Development. May 1, 2000; 127 (10): 2075-87.


Mouse Gli1 mutants are viable but have defects in SHH signaling in combination with a Gli2 mutation., Park HL., Development. April 1, 2000; 127 (8): 1593-605.


Regulation of Gli2 and Gli3 activities by an amino-terminal repression domain: implication of Gli2 and Gli3 as primary mediators of Shh signaling., Sasaki H., Development. September 1, 1999; 126 (17): 3915-24.


Essential function of Gli2 and Gli3 in the formation of lung, trachea and oesophagus., Motoyama J., Nat Genet. September 1, 1998; 20 (1): 54-7.


Diminished Sonic hedgehog signaling and lack of floor plate differentiation in Gli2 mutant mice., Ding Q., Development. July 1, 1998; 125 (14): 2533-43.


Combinatorial Gli gene function in floor plate and neuronal inductions by Sonic hedgehog., Ruiz i Altaba A., Development. June 1, 1998; 125 (12): 2203-12.


Evidence for the involvement of the Gli gene family in embryonic mouse lung development., Grindley JC., Dev Biol. August 15, 1997; 188 (2): 337-48.


Gli1 is a target of Sonic hedgehog that induces ventral neural tube development., Lee J., Development. July 1, 1997; 124 (13): 2537-52.                  


A binding site for Gli proteins is essential for HNF-3beta floor plate enhancer activity in transgenics and can respond to Shh in vitro., Sasaki H., Development. April 1, 1997; 124 (7): 1313-22.


Cloning and sequencing of the mouse Gli2 gene: localization to the Dominant hemimelia critical region., Hughes DC., Genomics. January 15, 1997; 39 (2): 205-15.


Specific and redundant functions of Gli2 and Gli3 zinc finger genes in skeletal patterning and development., Mo R., Development. January 1, 1997; 124 (1): 113-23.

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