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Summary Anatomy Item Literature (8703) Expression Attributions Wiki
XB-ANAT-506

Papers associated with embryonic structure (and abcc8)

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Xenopus as a model system for studying pancreatic development and diabetes., Kofent J., Semin Cell Dev Biol. March 1, 2016; 51 106-16.  


Recessive mutations in PCBD1 cause a new type of early-onset diabetes., Simaite D., Diabetes. October 1, 2014; 63 (10): 3557-64.


A universally conserved residue in the SUR1 subunit of the KATP channel is essential for translating nucleotide binding at SUR1 into channel opening., de Wet H., J Physiol. October 15, 2012; 590 (20): 5025-36.            


Homeoprotein hhex-induced conversion of intestinal to ventral pancreatic precursors results in the formation of giant pancreata in Xenopus embryos., Zhao H., Proc Natl Acad Sci U S A. May 29, 2012; 109 (22): 8594-9.                              


Transient expression of Ngn3 in Xenopus endoderm promotes early and ectopic development of pancreatic beta and delta cells., Oropeza D., Genesis. March 1, 2012; 50 (3): 271-85.                        


Neurally Derived Tissues in Xenopus laevis Embryos Exhibit a Consistent Bioelectrical Left-Right Asymmetry., Pai VP., Stem Cells Int. January 1, 2012; 2012 353491.          


Mutations of the same conserved glutamate residue in NBD2 of the sulfonylurea receptor 1 subunit of the KATP channel can result in either hyperinsulinism or neonatal diabetes., Männikkö R., Diabetes. June 1, 2011; 60 (6): 1813-22.              


The ATP-sensitive K(+)-channel (K(ATP)) controls early left-right patterning in Xenopus and chick embryos., Aw S., Dev Biol. October 1, 2010; 346 (1): 39-53.        


Activation of the K(ATP) channel by Mg-nucleotide interaction with SUR1., Proks P., J Gen Physiol. October 1, 2010; 136 (4): 389-405.                    


Xenopus pancreas development., Pearl EJ., Dev Dyn. June 1, 2009; 238 (6): 1271-86.          


A Kir6.2 mutation causing severe functional effects in vitro produces neonatal diabetes without the expected neurological complications., Tammaro P., Diabetologia. May 1, 2008; 51 (5): 802-10.            


Expression of complement components coincides with early patterning and organogenesis in Xenopus laevis., McLin VA., Int J Dev Biol. January 1, 2008; 52 (8): 1123-33.                                              


Functional effects of naturally occurring KCNJ11 mutations causing neonatal diabetes on cloned cardiac KATP channels., Tammaro P., J Physiol. February 15, 2006; 571 (Pt 1): 3-14.


The Mix family homeodomain gene bonnie and clyde functions with other components of the Nodal signaling pathway to regulate neural patterning in zebrafish., Trinh LA., Development. October 1, 2003; 130 (20): 4989-98.


K(ATP) channel activity is required for hatching in Xenopus embryos., Cheng SM., Dev Dyn. December 1, 2002; 225 (4): 588-91.        


Open state destabilization by ATP occupancy is mechanism speeding burst exit underlying KATP channel inhibition by ATP., Li L., J Gen Physiol. January 1, 2002; 119 (1): 105-16.                    


Amiloride derivatives are potent blockers of KATP channels., Bollensdorff C., Naunyn Schmiedebergs Arch Pharmacol. October 1, 2001; 364 (4): 351-8.


Chromanol 293B, a blocker of the slow delayed rectifier K+ current (IKs), inhibits the CFTR Cl- current., Bachmann A., Naunyn Schmiedebergs Arch Pharmacol. June 1, 2001; 363 (6): 590-6.


Glimepiride block of cloned beta-cell, cardiac and smooth muscle K(ATP) channels., Song DK., Br J Pharmacol. May 1, 2001; 133 (1): 193-9.


Effects of mitiglinide (S 21403) on Kir6.2/SUR1, Kir6.2/SUR2A and Kir6.2/SUR2B types of ATP-sensitive potassium channel., Reimann F., Br J Pharmacol. April 1, 2001; 132 (7): 1542-8.


Fast1 is required for the development of dorsal axial structures in zebrafish., Sirotkin HI., Curr Biol. September 7, 2000; 10 (17): 1051-4.


Nucleotide modulation of pinacidil stimulation of the cloned K(ATP) channel Kir6.2/SUR2A., Gribble FM., Mol Pharmacol. June 1, 2000; 57 (6): 1256-61.


Rat homolog of sulfonylurea receptor 2B determines glibenclamide sensitivity of ROMK2 in Xenopus laevis oocyte., Tanemoto M., Am J Physiol Renal Physiol. April 1, 2000; 278 (4): F659-66.


Altered functional properties of KATP channel conferred by a novel splice variant of SUR1., Sakura H., J Physiol. December 1, 1999; 521 Pt 2 337-50.


Pharmacological plasticity of cardiac ATP-sensitive potassium channels toward diazoxide revealed by ADP., D'hahan N., Proc Natl Acad Sci U S A. October 12, 1999; 96 (21): 12162-7.


Differential sensitivity of beta-cell and extrapancreatic K(ATP) channels to gliclazide., Gribble FM., Diabetologia. July 1, 1999; 42 (7): 845-8.


Identification of the high-affinity tolbutamide site on the SUR1 subunit of the K(ATP) channel., Ashfield R., Diabetes. June 1, 1999; 48 (6): 1341-7.


Tissue specificity of sulfonylureas: studies on cloned cardiac and beta-cell K(ATP) channels., Gribble FM., Diabetes. September 1, 1998; 47 (9): 1412-8.


Cloning and characterization of a novel human inwardly rectifying potassium channel predominantly expressed in small intestine., Partiseti M., FEBS Lett. August 28, 1998; 434 (1-2): 171-6.


Phentolamine block of KATP channels is mediated by Kir6.2., Proks P., Proc Natl Acad Sci U S A. October 14, 1997; 94 (21): 11716-20.


Expression of Xenopus snail in mesoderm and prospective neural fold ectoderm., Essex LJ., Dev Dyn. October 1, 1993; 198 (2): 108-22.              

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